METHOD FOR CHANGING BODY COLOR OF CRAWFISH, FEEDSTUFF TO BE FED AND CRAWFISH

• 1. 小食蚁兽饲料及其制备方法和用其饲养小食蚁兽的方法 [P] . 中国专利： CN114128794A . 2022-03-04
• 2. 桃小食心虫幼虫的饲料饲养装置及饲料饲养方法 [P] . 中国专利： CN111226873B . 2021.11.16
• 3. A manufacturing method thereof, and color reversible change dimmer and color reversible change display device and color reversible change display device for electrode body [P] . 日本专利： JP5169216B2 . 2013-03-27

  机译： 及其制造方法，电极体的颜色可逆变化调光器，颜色可逆变化显示装置和颜色可逆变化显示装置

• 4. A manufacturing method thereof, and color reversible change dimmer and color reversible change display device and color reversible change display device for electrode body [P] . 日本专利： JPWO2007020945A1 . 2009-02-26

  机译： 一种其制造方法，以及用于电极体的颜色可逆变化调光器和颜色可逆变化显示装置以及颜色可逆变化显示装置

• 5. COLOR-CHANGING DOOR BODY, REFRIGERATOR HAVING SAME, AND CONTROL METHOD FOR COLOR-CHANGING DOOR BODY [P] . WO2021212934A1 . 2021-10-28

  机译： 变色门体，具有相同的冰箱，以及变形门体的控制方法

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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Modeling Method of Automotive Body CAN/LIN Nets Application Protocol Based on Object-oriented Colored Petri Net [J] . FANG Hua ,HAN Jianghong ,LIU Xiaoping . 中国机械工程学报 . 2011,第006期
• 3. 冀北地区苹果桃小食心虫发生规律及防治方法研究 [C] . 姚明辉 ,李树才 . 中国植物保护学会2010年学术年会 . 2010
• 4. 桃小食心虫饲养方法优化及滞育激素基因表达模式研究 [A] . 林乾坤 . 2019