Neutralization processing method and ion chromatograph analysis, neutralization processor, and ion chromatograph device

• 1. 一种基于电中和再生的离子交换树脂的废水处理方法 [P] . 中国专利： CN112978857A . 2021-06-18
• 2. 低压离子色谱分析中所用抑制柱的自动再生装置 [P] . 中国专利： CN102631797B . 2015.02.18
• 3. Data processing device for distillation gas chromatograph analysis [P] . 日本专利： JP6148468B2 . 2017-06-14

  机译： 蒸馏气相色谱分析数据处理装置

• 4. Processing liquid chromatograph for extraction of small molecules from solid matrices and the method of chromatographic analysis using said equipment. [P] . BR102015021047A2 . 2017-03-07

  机译： 处理从固体基质中提取小分子的液相色谱仪和使用所述设备的色谱分析方法。

• 5. SUBSTRATE PROCESSING DEVICE, METHOD OF ADJUSTING PRESSURE IN SUBSTRATE PROCESSING DEVICE, AND METHOD OF EXECUTING CHARGE NEUTRALIZATION PROCESSING ON MOUNTING TABLE OF SUBSTRATE PROCESSING DEVICE [P] . 美国专利： US2008069669A1 . 2008-03-20

  机译： 基板处理装置，基板处理装置中的压力调整方法以及在基板处理装置的安装台上执行电荷中和处理的方法

• 1. Regime Map of Dry Neutralization Agglomeration Process Containing Non-Reactive Ingredient on a Laboratory Scale [J] . Leping Dang, Xu Li, Jinsheng Fu, 天津大学学报（英文版） . 2019,第001期
• 2. Nonlinear Model Algorithmic Control of a pH Neutralization Process [J] . ZOU Zhiyun, YU Meng, WANG Zhizhen, 中国化学工程学报（英文版） . 2013,第004期
• 3. Implementing Laboratory Method Development and Data Processing Software in Process Gas Chromatographs [C] . Christoph Klawun, Soheil Saadat Annual ISA Analysis Division symposium . 1999
• 4. Design, construction, and applications of an automated microprocessor-controlled gas chromatograph fraction collector-olfactometer. [D] . Salinas, Juan Pablo. 2000
• 5. Production of Cytomegalovirus Dense Bodies by Scalable Bioprocess Methods Maintains Immunogenicity and Improves Neutralizing Antibody Titers [O] . Kirsten Schneider-Ohrum, Corinne Cayatte, Yi Liu, 2016
• 6. Qualification of the First ICS-3000 ION Chromatograph for use at the Defense Waste Processing Facility [O] . Edwards, T, Mahannah, R. 2011
• 7. Class II Precision and Accuracy Study of the Analysis of VX in the Alternative Technologies and Approaches Program's VX/Caustic Neutralization Product Using a Gas Chromatograph/Ion Trap Mass Spectrometer [R] . Ellzy, M. W. 1998

• 1. APPLICATION OF CATIONIC STARCH BY DRY-PROCESS AS ANIONIC CHARGE NEUTRALIZING AGENTS TO IMPROVE FILLER RETENTION [J] . Fushan Chen ,Qijie Chen ,Gaosheng Wang . 天津科技大学学报 . 2004,第0z1期
• 2. APPLICATION OF CATIONIC STARCH BY DRY-PROCESS AS ANIONIC CHARGE NEUTRALIZING AGENTS TO IMPROVE FILLER RETENTION [J] . FushanChen ,QijieChen ,GaoshengWang . 天津科技大学学报 . 2004,第A01期
• 3. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 4. 使用自动中和装置－离子色谱联用技术检测浓酸中的痕量阳离子 [C] . 刘立洋 ,刘肖 . 第13届离子色谱学术报告会 . 2010
• 5. 基于电渗析原理的样品中和、氧化、萃取前处理方法的研究及其在离子色谱中的应用 [A] . 胡康康 . 2009