Method for judging collision with three directional accelerative signals and apparatus for performing the same

• 1. 执行判断装置、接收装置、无线通信系统和执行判断方法 [P] . 中国专利： CN101933249A . 2010-12-29
• 2. 日程活动冲突判断方法、电子设备及存储介质 [P] . 中国专利： CN113487272A . 2021-10-08
• 3. METHOD FOR JUDGING COLLISION WITH THREE DIRECTIONAL ACCELERATIVE SIGNALS AND APPARATUS FOR PERFORMING THE SAME [P] . IN1995MA01399A . 2010-03-26

  机译： 带有三个方向性加速度信号的碰撞判断方法和执行该方法的装置

• 4. Method for judging collision with three directional accelerative signals and apparatus for performing the same [P] . 美国专利： US5732374A . 1998-03-24

  机译： 判断与三个方向性加速度信号的冲突的方法和执行该方法的设备

• 5. Method for judging collision with three directional accelerative signals and apparatus for performing the same [P] . 欧洲知识产权局专利： EP0709256B1 . 1997-09-03

  机译： 判断与三个方向性加速度信号的冲突的方法和执行该方法的设备

• 1. BRMS1 performs an anti-tumor effect on renal cell carcinoma via inhibition of PI3K/AKT/mTOR signaling pathway [J] . Ya-nan Cong, Zhen-guo Wang, Lin Zhao 海南医科大学学报（英文版） . 2019,第016期
• 2. BRMS1 performs an anti-tumor effect on renal cell carcinoma via inhibition of PI3K/AKT/mTOR signaling pathway [J] . Ya-nan Cong, Zhen-guo Wang, Lin Zhao 海南医科大学学报(英文版) . 2019,第016期
• 3. Study on Measurement Methods of Multi-Directional Waves-Wave Gauge Array Method [J] . 中国海洋工程（英文版） . 1992,第002期
• 4. Development of Intention Inference Algorithm Based on EMG Signals at Judging Directional of Arrow Cues [C] . Yuzo Takahashi International Ergonomics Association, Congress . 2018
• 5. Methods, devices, and apparatus for performing a vascular anastomosis [D] . Gehrke, Cody Lee 2012
• 6. A Rapid Method of Performing Enterectomy without the Aid of any Special Apparatus [O] . Leonard Rogers 1896
• 7. GPU accelerated Nature Inspired Methods for Modelling Large Scale Bi-Directional Pedestrian Movement [O] . Dutta, Sankha Baran, McLeod, Robert, Friesen, Marcia 2014
• 8. Empirical mode decomposition apparatus, method and article of manufacture for analyzing biological signals and performing curve fitting [R] . 2004

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. The mechanism study of glycosides of cistanches improv ing cognitive performance dysfunction in senescence accelerated mouse prone 8 by AMPK/mTOR signaling pathway [J] . Liu Xinlang ,Zhou Lili ,Sun Yihong . 解剖学杂志 . 2021,第S01期
• 3. LAMOST光纤定位单元冲突的判断方法 [C] . 杨飞 ,中国科学院国家天文台LAMOST项目组 ,王钢 . 2004第四届精密工程学术研讨会 . 2004
• 4. 基于加速度信号的下肢运动功能评价方法 [A] . 孙纯岭 . 2019