process and device for the operation of a filter press with a lung air in references: have built downstream

• 1. 用于安装在移动装置中的钥匙开关以及用于操作该钥匙开关的方法 [P] . 中国专利： CN101874258B . 2012.09.26
• 2. 用于安装在移动装置中的钥匙开关以及用于操作该钥匙开关的方法 [P] . 中国专利： CN101874258A . 2010-10-27
• 3. Method and apparatus for the operation of a filter press with a downstream air vessel [P] . 德国专利： DE2548842C3 . 1980-03-13

  机译： 用于具有下游空气容器的压滤机的操作方法和设备

• 4. Method and apparatus for the operation of a filter press with a downstream air vessel [P] . 德国专利： DE2548842B2 . 1979-07-12

  机译： 用于具有下游空气容器的压滤机的操作方法和设备

• 5. Method and apparatus for the operation of a filter press with a downstream air vessel [P] . 德国专利： DE2548842A1 . 1977-05-05

  机译： 用于具有下游空气容器的压滤机的操作方法和设备

• 1. Effect of the adaptive intermittent ventilation before radical operation for lung cancer under one-lung ventilation on the non-ventilated lung tissue injury and apoptosis molecule protein expression [J] . Xue-Mei Yang 海南医科大学学报（英文版） . 2017,第018期
• 2. Effect of the adaptive intermittent ventilation before radical operation for lung cancer under one-lung ventilation on the non-ventilated lung tissue injury and apoptosis molecule protein expression [J] . Xue-Mei Yang 海南医科大学学报(英文版) . 2017,第018期
• 3. Interference Suppression for MIMO Device to Device Pairs Underlaying Cellular Network [J] . WANG Huan, LI Li, WANG Bin, 东华大学学报（英文版） . 2017,第005期
• 4. Resveratrol inhibits the P38MAPK pathway as well as downstream apoptosis, inflammation and oxidative stress molecule expression in secondary lung injury in intestinal ischemia-reperfusion [J] . Xiao-Guang Wang, Xiao-Fei Qi, Ge-Hui Li 海南医科大学学报（英文版） . 2017,第012期
• 5. Simultaneous Synthesis of the Downstream Process and the Reactor Concept for the Oxidative Coupling of Methane (OCM) Steffen Stuenkel, Hamid R. Godini, Jens-Uwe Repke, Guenter Wozny Berlin Institute of Technology, Department of Process Engineering, Chair of Process Operation and Dynamics, Strafie des 17. Juni 135, 10623 Berlin, GermanyMSteffen.Stuenkel@tu-berlin.de [C] . International Symposium on Process Systems Engineering . 2009
• 6. A study of ligand-induced erbB receptor interactions and downstream signaling in an erbB-2 overexpression model for lung cancer [D] . Fernandes, Audrey Maria 1998
• 7. Low suction on digital drainage devices promptly improves post-operative air leaks following lung resection operations: a retrospective study [O] . Suguru Mitsui, Shunsuke Tauchi, Takahiro Uchida, 2021
• 8. Extraction of Device Fingerprints Using Built-in Erase-Suspend Operation of Flash Memory Devices [O] . The-Nghia Nguyen, Sunghyun Park, Donghwa Shin 2020

• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. GB/T21087-2007《空气-空气能量通风装置》附录E.2中有关全热交换器测试方法的探讨 [C] . . 第十三届全国热泵与系统节能技术大会 . 2008
• 3. 国产压滤机在精对苯二甲酸装置中的应用研究 [A] . 孙中田 . 2014