Process for the treatment of continuous waste water streams having changing contents of different oxidizable materials with hydrogen peroxide

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• 3. PROCESS FOR THE CONTINUOUS TREATMENT WITH HYDROGEN PEROXIDE OF WASTE WATER STREAMS HAVING VARIABLE CONTENTS OF OXIDIZABLE SUBSTANCES [P] . IL66128A . 1985-11-29

  机译： 具有多种可氧化物质含量的废水水流中过氧化氢的连续处理方法

• 4. PROCESS FOR THE HYDROGEN PEROXIDE TREATMENT OF CONTINUOUS STREAMS/FLOWS/CURRENTS OF WASTE WATER HAVING VARYING CONTENTS OF DIFFERENT OXIDIZABLE COMPONENTS [P] . ZA824426B . 1983-07-27

  机译： 具有不同可氧化成分含量的废水连续流/流/流过氧化氢处理方法

• 5. PROCESS FOR THE CONTINUOUS TREATMENT WITH HYDROGEN PEROXIDE OF WASTE WATER STREAMS HAVING VARIABLE CONTENTS OF OXIDIZABLE SUBSTANCES [P] . IL66128D0 . 1982-09-30

  机译： 具有多种可氧化物质含量的废水水流中过氧化氢的连续处理方法

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• 6. Hydrogen Peroxide and Peracetic Acid Oxidizing Potential in the Treatment of Water [O] . Elena Madalina Popescu, Octav Pantea, Daniela Gologan, 2019
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• 1. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 2. Separating trash and solid materials from the wastewater ammonia stripping tower UASB biological oxidation pond processes for the treatment of the swine factory＇s wastewater [J] . LU Xiu-guo ,ZHANG Pan ,RAO Ting . 环境科学与工程 . 2009,第003期
• 3. Degradation of 2,4-dichlorophenoxyacetic acid in water by ozonehydrogen peroxide process [J] . YU Ying-hui MA Jun HOU Yan-jun . 环境科学学报：英文版 . 2006,第6期
• 4. 用过氧化氢处理硫酸盐厂的臭气问题 [C] . 劳嘉葆 . 2001中国造纸化学品开发应用国际技术交流会 . 2001
• 5. 废新闻纸回用过程中产生的溶解与胶体物质的研究 [A] . 李学凤 . 2008