METHOD FOR DETECTING CONTENT OF TOXIC SUBSTANCES IN EXHAUST GASES OF INTERNAL COMBUSTION ENGINE OF VEHICLE

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  机译： 燃料添加剂用于减少内燃机废气中有毒物质含量的用途

• 4. A method for reducing the content of the exhaust gases of internal combustion engines to incombustible, in particular toxic constituents [P] . 德国专利： DE1476193A1 . 1969-02-06

  机译： 一种将内燃机的废气含量减少到不可燃的，特别是有毒成分的方法

• 5. A method for reducing the content of harmful substances in the exhaust gases of internal combustion engines or - motors and apparatus for carrying out the method [P] . 德国专利： DE1815834A1 . 1969-07-24

  机译： 减少内燃机或发动机排气中有害物质含量的方法和实施该方法的电动机和装置

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• 1. Comparative Analysis of Velocity Decomposition Methods for Internal Combustion Engines [J] . Semih Olcmen ,Marcus Ashford ,Philip Schinestsky . 流体动力学（英文） . 2012,第3期
• 2. STEADY-STATE AND IDLE OPTIMIZA- TION OF INTERNAL COMBUSTION ENGINE CONTROL STRATEGIES FOR HYBRID ELECTRIC VEHICLES [J] . WANG Feng ,MAO Xiaojian ,YANG Lin . 中国机械工程学报 . 2008,第002期
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• 4. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 5. Effect of iγ/i-Valerolactone Blending on Engine Performance, Combustion Characteristics and Exhaust Emissions in a Diesel Engine [J] . ákos Bereczky ,Kristóf Lukács ,Mária Farkas . 自然资源（英文） . 2014,第5期
• 6. 内燃机排气成份动态检测方法的研究 [C] . . 中国汽车工程学会发动机分会.中国内燃机学会汽油机煤气机分会2000年度学术年会 . 2000
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