DEVICE AND METHOD FOR REVERSIBLY CONNECTING A REHABILITATION MECHANISM TO A BED, AND METHOD FOR OPERATING A REHABILITATION MECHANISM CONNECTED TO A BED

• 1. 一种用于将康复机构可逆地连接到床的装置和方法，以及用于操作连接到床的康复机构的方法 [P] . 中国专利： CN113271906A . 2021-08-17
• 2. 用于将浇斗护罩可逆地联接到收集器喷嘴的联接装置、自支撑浇斗护罩、其套件以及用于将浇斗护罩联接到收集器喷嘴的方法 [P] . 中国专利： CN105636723B . 2018.01.02
• 3. DEVICE AND METHOD FOR REVERSIBLY CONNECTING A REHABILITATION MECHANISM TO A BED, AND METHOD FOR OPERATING A REHABILITATION MECHANISM CONNECTED TO A BED [P] . EP3883516A1 . 2021-09-29

  机译： 可逆地将康复机构可逆地连接到床的装置和方法以及操作连接到床的康复机构的方法

• 4. DEVICE AND METHOD FOR REVERSIBLY CONNECTING A REHABILITATION MECHANISM TO A BED, AND METHOD FOR OPERATING A REHABILITATION MECHANISM CONNECTED TO A BED [P] . 世界知识产权组织专利： WO2020103983A1 . 2020-05-28

  机译： 用于将修复机制可逆地连接到床的装置和方法，以及用于操作连接到病床的修复机制的方法

• 5. Apparatus for reversibly connecting a rehabilitation device to a bed and a method for operating a rehabilitation device connected to the bed [P] . KR20210093277A . 2021-07-27

  机译： 用于可逆地将康复装置连接到床的床和用于操作连接到床的康复装置的方法

• 1. Penile rehabilitation with a vacuum erectile device in an animal model is related to an antihypoxic mechanism:blood gas evidence [J] . Hao-Cheng Lin, Wen-Li Yang, Jun-Lan Zhang, 亚洲男性学杂志（英文版） . 2013,第003期
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• 4. Optimal Design of Lifting Mechanism for Standing Rehabilitation Bed based on MATLAB [C] . Guimin Wu, Xiang Shi International Conference on Mechanical and Electronics Engineering . 2013
• 5. A Method to Enhance the Heat Transfer Mechanism in Metal Particle Porous Beds with Applicability to Paramagnetic Materials for Hydrogen Storage. [D] . Shafiee, Shahin. 2016
• 6. Experiment and Mechanism Analysis of Desulfurizationwith a MgO-Based Desulfurizer Fixed Bed Method [O] . Wan Cao, WeiJun Zhang, * 2020
• 7. Mechanism design and kinematics analysis of multifunctional waist rehabilitation bed [O] . Liang Liu, Jingjing Ma, YanYing Luo, 2019

• 1. Research on Combined Rehabilitation Method and Its Mechanism of Traditional Chinese Medicine Daoyin Technique with Biofeedback Technique [J] . Wenru Zhao ,Pengchuan Zhang ,Ming Qi . 生物医学工程（英文） . 2019,第12期
• 2. BACKGROUND： Vibrio vulnifi cus inside the body could activate the NF-!B signaling pathwayand initiate the inflammatory cascade. The lung is one of the earliest organs affected by sepsisassociated with acute lung injury. High mobility group protein B1 （HMGB1） is an important late-actingpro-infl ammatory cytokine involving in the pathophysiology of sepsis. It is also involved in the injuryprocess in the lung, liver and intestine. There has been no report on the involvement of HMGB1 inVibrio vulnifi cus sepsis-induced lung injury.METHODS： Sixty rats were randomly divided into a normal control group （group A, n=10） anda Vibrio vulnificus sepsis group （group B, n=50）. Sepsis was induced in the rats by subcutaneousinjection of Vibrio vulnificus （concentration 6×108 cfu/mL, volume 0.1 mL/100g）） into the left lowerlimbs. The rats in group B were sacrifi ced separately 1, 6, 12, 24, and 48 hours after the infection.Their lungs were stored as specimens, lung water content was measured, and lung pathology wasobserved under a light microscope. The expressions of the HMGB1 gene and protein in the lungswere detected by RT-PCR and Western blot. Data were analyzed with one-way analysis of variance（ANOVA） and the LSD method for pair-wise comparison between the two groups. P〈0.05 wasconsidered statistically signifi cant.RESULTS： Compared to group A （0.652±0.177）, HMGB1 mRNA expression in the lungs ofgroup B was signifi cantly higher at 0 hour （1.161±0.358, P=0.013）, 24 hours （1.679±0.235, P=0.000）,and 48 hours （1.258±0.274, P=0.004） （P〈0.05）, and peaked at 24 hours. Compared to group A（0.594±0.190）, HMGB1 protein expression at 6 hours （1.408±0.567, P=0.026） after infection wassignificantly increased （P〈0. 05）, and peaked at 24 hours （2.415±1.064, P=0.000） after infection.Compared to group A （0.699±0.054）, lung water content was significantly increased at 6 hours（0.759±0.030, P=0.001）,12 hours （0.767±0.023, P=0.000）, 24 hours （0.771±0.043, P=0.000） and 48hours （0.789±0.137, P=0.000） after infection （P〈0.05）. Compared to group A, pathological changesat 12 hours in group B indicate marked pulmonary vascular congestion, interstitial edema andinfl ammatory infi ltration. Alveolar cavity collapse and boundaries of the alveolar septum could not beclearly identifi ed.CONCLUSION： Vibrio vulnifi cus sepsis can lead to injury in rat lungs, and increased HMGB1expression in lung tissue may be one of the mechanisms for injury from Vibrio vulnifi cus sepsis. [J] . Qiao-meng Qiu ,Zhong-wang Li ,Lu-ming Tang . 世界急诊医学杂志(英文) . 2011,第4期
• 3. V法铸造工艺的新发展——将消失模工艺嫁接到V法铸造工艺的新方法 [C] . 马怀荣 ,刘祥泉 ,韩建华 . 第十届全国消失模与V法铸造学术年会 . 2011
• 4. 床式下肢康复训练机器人控制方法研究 [A] . 刘波 . 2017