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首页> 外文会议>Conference on Laser and Noncoherent Light Ocular Effects: Epidemiology, Prevention, and Treatment III Jan 26-27, 2003 San Jose, California, USA >A Foveal Photoreceptor Explanation of Short Term Visual Acuity Recovery Associated with Laser Induced Foveal Damage
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A Foveal Photoreceptor Explanation of Short Term Visual Acuity Recovery Associated with Laser Induced Foveal Damage

机译：与激光诱发的中央凹损伤相关的短期视敏度恢复的中央凹感光器解释。

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Both human laser accident cases and non-human primate behavioral studies demonstrate the possibility of full visual acuity recovery following foveal laser injury. Current explanations of such recovery require suppositions of complex retinal reorganization dynamics or neural reorganization at higher order visual brain systems. However, recent investigation based on data of retinal photoreceptor and ganglion cell topography and connectivity, suggest that the amount of static inherent plasticity, already exists at the retinal level, may also explain visual acuity recovery in the presence of laser-induced foveal damage. Modeling the off-axis visual acuity while utilizing this data, produces a more gradual fall-off in visual acuity, and supports the notion that visual acuity recovery may reside in the topographical organization of the cones. Moreover, considering the filling-in phenomena, which can conceal the presence of retinal damage from being recognized, together with eye movements, could nullify scotoma, as long as the retinal damage is not too extensive.
机译：人类激光事故案例和非人类灵长类动物行为研究均表明，中央凹激光损伤后完全视力恢复的可能性。当前关于这种恢复的解释要求在较高阶的视觉脑系统中假设复杂的视网膜重组动力学或神经重组。但是，最近基于视网膜感光器和神经节细胞的地形和连接性数据进行的研究表明，在视网膜水平上已经存在的静态固有可塑性数量，也可以解释在激光诱发的中央凹损伤时视力恢复的情况。在利用此数据进行轴外视敏度建模时，会导致视敏度逐渐下降，并支持视敏度恢复可能驻留在视锥的地形组织中的观点。而且，考虑到可以掩盖视网膜损害的存在以及眼睛运动的填充现象，只要视网膜损害不是太广泛，就可以使暗影消失。

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