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Abnormal passive and intrinsic stiffness in the spastic ankle

机译：痉挛脚踝异常被动和固有刚度

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A parallel-cascade system identification method was used to measure dynamic ankle stiffness in normal and spastic spinal cord injured (SCI) subjects. Modulation of passive and intrinsic stiffness gain of ankle extensor muscles (GS) was studied by applying perturbations to the ankle at different positions under passive (relaxed) and active (10% extensor maximum voluntary contraction) conditions. Both passive and intrinsic stiffness were described well by a linear second-order model having elastic, viscous and inertia parameters. For passive stiffness the elastic and viscous parameters increased with ankle dorsiflexion in both groups; in addition, both parameters were larger in SCI than in control subjects as ankle dorsiflexed. For intrinsic stiffness (1) the elastic parameter increased with ankle position until neutral position (90/spl deg/) in both groups, it then saturated in control subjects, but decreased as ankle moved to dorsiflexion in SCI subjects; (2) it was always smaller in SCI subjects; (3) the intrinsic viscous parameter increased at the beginning of plantarflexion then decreased slightly until maximum dorsiflexion in both groups. However, it was lower than in control subjects as the ankle moved from neutral position to maximum dorsiflexion. The results indicate that abnormal non-reflex mechanics are due to enhanced passive stiffness and reduced intrinsic stiffness.
机译：使用平行级联系统识别方法测量正常和痉挛性脊髓受损（SCI）受试者的动态踝僵硬度。通过在被动（松弛）下的不同位置处的踝关节和有源（10％伸展最大自愿收缩）条件下，研究了踝关节伸肌肌肉（GS）的被动和固有刚度增益的调制。通过具有弹性，粘性和惯性参数的线性二阶模型很好地描述了被动和内在刚度。对于被动刚度，弹性和粘性参数随着两个组中的踝背裂增加而增加;此外，SCI中的两个参数比控制受试者更大，因为脚踝背屈。对于固有刚度（1），弹性参数随踝部位置的增加而增加，直到两个组中的中性位置（90 / SPL次/），然后在对照受试者中饱和，但随着踝关节移动到SCI受试者中的背裂，因此减少; （2）SCI科目总是更小; （3）在Plantarflexion开始时的固有粘性参数随后略微降低，直到两组中的最大背屈。然而，当脚踝从中性位置移动到最大背裂时，它低于控制受试者。结果表明，异常的非反射力学是由于增强的被动刚度和内在刚度降低。

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