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Caffeic Acid Phenethyl Ester Protect Mice Hepatic Damage Against Cadmium Exposure

机译：咖啡酸苯乙烯酯保护小鼠肝脏损伤对镉暴露

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The aim of this study is to approach the possible effect and mechanism of caffeic acid phenethyl ester (CAPE) against the toxicity of Cadmium(Cd) on mice liver. Methods: In vivo assays have been performed by evaluating the oxidative damage marker and the antioxidant parameters, meanwhile, the histopathological changes were also observed. Results: It was revealed that the intoxication of Cd could increase the concentration of oxidative damage and decrease the content of antioxidant system, the impact of Cd on liver damage can be suppressed by the effect of CAPE by lowering the oxidative damage levels and improving the activities of antioxidant enzymes. Conclusion: From the data obtained from the studies, we found that the protection effect of CAPE on Cd toxicity was not solely dependent on the antioxidant effect, however, the evidence of CAPE playing a role in the amelioration of Cd toxicity probably showed that CAPE can be a promising agent in the treatment of Cd exposure.
机译：本研究的目的是接近咖啡酸苯乙烷酯（辣椒）对小鼠肝脏镉（CD）毒性的可能效果和机制。方法：通过评估氧化损伤标记和抗氧化剂参数，对体内测定进行了同时，同时也观察到组织病理学变化。结果：揭示了CD的中毒可以增加氧化损伤的浓度并降低抗氧化系统的含量，CD对肝脏损伤对肝脏损伤的影响可以通过降低氧化损伤水平并改善活动来抑制辣椒的作用抗氧化酶。结论：从研究中获得的数据，我们发现斗篷对Cd毒性的保护作用并非仅仅依赖于抗氧化效果，然而，在CD毒性的改善中发挥作用的斗篷的证据可能表明斗篷可以是治疗CD暴露的有希望的剂。

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