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Preclinical imaging of the co-stimulatory molecules CD80 and CD86 with indium-111-labeled belatacept in atherosclerosis

机译：共刺激分子CD80和CD86与铟111标记的belatacept在动脉粥样硬化中的临床前成像

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摘要

BackgroundThe inflammatory nature of atherosclerosis provides a broad range of potential molecular targets for atherosclerosis imaging. Growing interest is focused on targets related to plaque vulnerability such as the co-stimulatory molecules CD80 and CD86. We investigated in this preclinical proof-of-concept study the applicability of the CD80/CD86-binding fusion protein belatacept as a probe for atherosclerosis imaging.

机译：背景动脉粥样硬化的炎症性质为动脉粥样硬化成像提供了广泛的潜在分子靶标。人们越来越关注与斑块易损性有关的靶标，例如共刺激分子CD80和CD86。我们在此临床前概念验证研究中调查了CD80 / CD86结合融合蛋白belatacept作为动脉粥样硬化成像探针的适用性。

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期刊名称 EJNMMI Research
作者
Romana Meletta; Adrienne Müller Herde; Patrick Dennler; Eliane Fischer; Roger Schibli; Stefanie D. Krämer;
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作者单位

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年(卷),期 2016(6),-1
年度 2016
页码 1
总页数 12
原文格式 PDF
正文语种
中图分类
关键词
Co-stimulatory molecules CD80 CD86 Belatacept Atherosclerosis Inflammation Imaging;

机译：共刺激分子;CD80;CD86;Belatacept;动脉粥样硬化;炎症;成像;

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专利

1. Preclinical imaging of the co-stimulatory molecules CD80 and CD86 with indium-111-labeled belatacept in atherosclerosis [J] . Romana Meletta, Adrienne Müller Herde, Patrick Dennler, EJNMMI Research . 2016,第Suppla1期

机译：共刺激分子CD80和CD86与铟111标记的belatacept在动脉粥样硬化中的临床前成像
2. Sex-Associated Expression of Co-Stimulatory Molecules CD80, CD86, and Accessory Molecules, PDL-1, PDL-2 and MHC-II, in F480+ Macrophages during Murine Cysticercosis [J] . Cristian Togno-Peirce, Karen Nava-Castro, Luis Ignacio Terrazas, BioMed research international . 2013,第Pta3期

机译：在鼠膀胱术期间，在F480 +巨噬细胞中的性刺激分子CD80，CD86和辅助分子，PDL-1，PDL-2和MHC-II中的性相关表达
3. Sex-Associated Expression of Co-Stimulatory Molecules CD80, CD86, and Accessory Molecules, PDL-1, PDL-2 and MHC-II, in F480+ Macrophages during Murine Cysticercosis [J] . CristiánTogno-Peirce, KarenNava-Castro, Luis IgnacioTerrazas, BioMed research international . 2013,第10期

机译：鼠囊性囊病期间F480 +巨噬细胞中共刺激分子CD80，CD86和辅助分子PDL-1，PDL-2和MHC-II的性别相关表达
4. Smaller sizes of Ag-PVP Nanoparticles Control Inflammatory Responses, and Reduce CD80 and CD86 Expression Levels, in Macrophages Infected with Chlamydia trachomatis [C] . Abebayehu N. Yilma, Shree R. Singh, Stacie J. Fairley, NSTI nanotechnology conference and expo;Nanotech conference expo;NSTI-Nanotech 2012;TechConnect world annual conference expo . 2012

机译：在感染了沙眼衣原体的巨噬细胞中，较小尺寸的Ag-PVP纳米颗粒可控制炎症反应并降低CD80和CD86表达水平。
5. Signaling through CD28 family receptors: An analysis of CD80- and CD86-dependent pathways. [D] . Slavik, Jacqueline Marie. 1998

机译：通过CD28家族受体发出信号：对CD80和CD86依赖性途径的分析。
6. Sex-Associated Expression of Co-Stimulatory Molecules CD80, CD86, and Accessory Molecules, PDL-1, PDL-2 and MHC-II, in F480+ Macrophages during Murine Cysticercosis [O] . Cristián Togno-Peirce, Karen Nava-Castro, Luis Ignacio Terrazas, 2006

机译：鼠囊性囊病期间F480 +巨噬细胞中共刺激分子CD80，CD86和辅助分子PDL-1，PDL-2和MHC-II的性别相关表达
7. Preclinical imaging of the co-stimulatory molecules CD80 and CD86 with indium-111-labeled belatacept in atherosclerosis [O] . 2016

机译：共刺激分子CD80和CD86与铟111标记的belatacept在动脉粥样硬化中的临床前成像

Preclinical imaging of the co-stimulatory molecules CD80 and CD86 with indium-111-labeled belatacept in atherosclerosis

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