Human parainfluenza virus type 3 (HPIV3) induces production of IFNγ and RANTES in human nasal epithelial cells (HNECs)

机译：3型人副流感病毒（HPIV3）诱导人鼻上皮细胞（HNEC）产生IFNγ和RANTES

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BackgroundHuman parainfluenza virus type 3 (HPIV3), while infecting lower airway epithelial cells induces pneumonia and bronchiolitis in infants and children, and may lead to asthma exacerbations in children and adults. Respiratory viruses invading the airway epithelium activate innate immune response and induce inflammatory cytokine release contributing to the pathophysiology of upper and lower airway disorders. However, the effects of HPIV3 infection on nasal epithelial cells have not been well defined.The aim of this study was to evaluate the effect of the HPIV3 infection on cultured human nasal epithelial cells (HNECs) and the release of interferon gamma and other cytokines.

机译：背景3型人类副流感病毒（HPIV3）在感染下呼吸道上皮细胞的同时，会引起婴儿和儿童的肺炎和细支气管炎，并可能导致儿童和成人的哮喘加重。侵入气道上皮的呼吸道病毒激活先天免疫反应并诱导炎症性细胞因子释放，从而导致上，下气道疾病的病理生理。然而，HPIV3感染对鼻上皮细胞的影响尚未明确。本研究的目的是评估HPIV3感染对培养的人鼻上皮细胞（HNEC）的影响以及干扰素γ和其他细胞因子的释放。

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期刊名称 Journal of Inflammation (London England)
作者
Anna Lewandowska-Polak; Małgorzata Brauncajs; Edyta Paradowska; Marzanna Jarzębska; Marcin Kurowski; Sylwia Moskwa; Zbigniew J Leśnikowski; Marek L Kowalski;
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年(卷),期 2015(12),-1
年度 2015
页码 16
总页数 7
原文格式 PDF
正文语种
中图分类免疫学;
关键词
PIV3 Human parainfluenza virus 3 Nasal epithelium IFN-γ RANTES RPMI cells;

机译：PIV3;人副流感病毒3;鼻上皮;IFN-γ;RANTES;RPMI细胞;

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1. Human parainfluenza virus type 3 (HPIV3) induces production of IFNγ and RANTES in human nasal epithelial cells (HNECs) [J] . Anna Lewandowska-Polak, Ma?gorzata Brauncajs, Edyta Paradowska, Journal of inflammation. . 2015,第S1期

机译：3型人副流感病毒（HPIV3）诱导人鼻上皮细胞（HNEC）产生IFNγ和RANTES
2. Deletion of the D domain of the human parainfluenza virus type 3 (HPIV3) PD protein results in decreased viral RNA synthesis and beta interferon (IFN-β) expression [J] . RothJ.P., LiJ.K.-K., MorreyJ.D., Virus Genes . 2013,第1期

机译：删除人类副流感病毒3型（HPIV3）PD蛋白的D结构域会导致病毒RNA合成和β干扰素（IFN-β）表达下降
3. Human trachea primary epithelial cells express both sialyl(alpha 2-3)Gal receptor for human parainfluenza virus type 1 and avian influenza viruses, and sialyl(alpha 2-6)Gal receptor for human influenza viruses [J] . Kogure T, Suzuki T, Takahashi T, Glycoconjugate journal . 2006,第1a2期

机译：人气管原代上皮细胞同时表达人副流感病毒1型和禽流感病毒的唾液酸（alpha 2-3）Gal受体和人流感病毒的唾液酸（alpha 2-6）Gal受体
4. Cell-Type-Dependent Effect of Transforming Growth Factor-beta, a Major Cytokine in Breast Milk, on Human Immunodeficiency Virus Type 1 Infection of Mammary Epithelial MCF-7 Cells or Macrophages [C] . M. Moriuchi, H. Moriuchi International AIDS Conference . 2004

机译：转化生长因子-β，母乳中主要细胞因子的细胞型依赖性效应，对人免疫缺陷病毒1型感染乳腺上皮MCF-7细胞或巨噬细胞
5. Investigation of the mechanism by which the human papillomavirus type-16 *E6 oncoprotein induces telomerase in epithelial cells. [D] . Gewin, Lindy. 2004

机译：人乳头瘤病毒16型* E6癌蛋白诱导上皮细胞端粒酶机制的研究。
6. Role of Nucleolin in Human Parainfluenza Virus Type 3 Infection of Human Lung Epithelial Cells [O] . Santanu Bose, Mausumi Basu, Amiya K. Banerjee 2004

机译：核仁蛋白在人副流感病毒3型人肺上皮细胞感染中的作用
7. Human parainfluenza virus type 3 (HPIV3) induces production of IFNγ and RANTES in human nasal epithelial cells (HNECs) [O] . 2015

机译：3型人副流感病毒（HPIV3）诱导人鼻上皮细胞（HNEC）产生IFNγ和RANTES
8. Report on Carcinogens (RoC) Concept: Selected Viruses. Epstein--Barr Virus, Human ImmunodeficiencynVirus Type 1, Human T--Cell Lymphotropic Virus Type 1, Kaposi Sarcoma--Associated Herpes Virus, and nMerkel Cell Polyoma Virus. [R] . 2016

机译：关于致癌物（RoC）概念的报告：选定的病毒。爱泼斯坦 - 巴尔病毒，人类免疫缺陷病毒1型，人类T细胞淋巴细胞病毒1型，卡波西肉瘤 - 相关疱疹病毒和nmerkel细胞多瘤病毒。

Human parainfluenza virus type 3 (HPIV3) induces production of IFNγ and RANTES in human nasal epithelial cells (HNECs)

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