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Endothelial Nitric Oxide Synthase Plays an Obligatory Role in the Late Phase of Ischemic Preconditioning by Activating the Protein Kinase C∊–p44/42 Mitogen-Activated Protein Kinase–pSer-Signal Transducers and Activators of Transcription1/3 Pathway

机译：内皮一氧化氮合酶通过激活蛋白激酶C∊–p44 / 42丝裂原激活的蛋白激酶–pSer-信号转导子和转录1/3通路的激活子在缺血预处理的后期起强制性作用。

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BackgroundThe role of endothelial nitric oxide synthase (eNOS) in ischemic preconditioning (PC) and cardioprotection is poorly understood. We addressed this issue using a genetic, rather than pharmacological, approach.

机译：背景内皮一氧化氮合酶（eNOS）在缺血预处理（PC）和心脏保护中的作用了解甚少。我们使用遗传方法而非药理方法解决了这个问题。

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期刊名称 other
作者
Yu-Ting Xuan; Yiru Guo; Yanqing Zhu; Ou-Li Wang; Gregg Rokosh; Roberto Bolli;
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年(卷),期 -1(116),5
年度 -1
页码 535–544
总页数 21
原文格式 PDF
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关键词
ischemia myocardial infarction occlusion stress;

机译：缺血;心肌梗塞;阻塞;压力;

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专利

1. Hepatocyte growth factor stimulates nitric oxide production through endothelial nitric oxide synthase activation by the phosphoinositide 3-kinase/Akt pathway and possibly by mitogen-activated protein kinase kinase in vascular endothelial cells. [J] . Uruno A, Sugawara A, Kanatsuka H, Hypertension research: Official journal of the Japanese Society of Hypertension . 2004,第11期

机译：肝细胞生长因子通过磷酸肌醇3-激酶/ Akt途径以及可能通过血管内皮细胞中的促分裂原活化蛋白激酶激酶激活内皮一氧化氮合酶，从而刺激一氧化氮的产生。
2. Platelet-derived-growth-factor stimulation of the p42/p44 mitogen-activated protein kinase pathway in airway smooth muscle: role of pertussis-toxin-sensitive G-proteins, c-Src tyrosine kinases and phosphoinositide 3-kinase. [J] . Conway AM, Rakhit S, Pyne S, The Biochemical Journal . 1999,第Pta2期

机译：气道平滑肌中p42 / p44丝裂原激活的蛋白激酶途径的血小板衍生生长因子刺激：百日咳毒素敏感的G蛋白，c-Src酪氨酸激酶和磷酸肌醇3激酶的作用。
3. Platelet-derived-growth-factor stimulation of the p42/p44 mitogen-activated protein kinase pathway in airway smooth muscle: role of pertussis-toxin-sensitive G-proteins, c-Src tyrosine kinases and phosphoinositide 3-kinase [J] . Ann-Marie CONWAY, Nigel J. PYNE, Soma RAKHIT, The biochemical journal . 1999,第2期

机译：气道平滑肌中p42 / p44丝裂原活化蛋白激酶途径的血小板衍生生长因子刺激：百日咳毒素敏感的G蛋白，c-Src酪氨酸激酶和磷酸肌醇3激酶的作用
4. Activation of Mitogen-activated Protein Kinases and Protein Kinase B/akt Signaling By Oxidative Stress in Vascular Smooth Muscle Cells: involvement in Vascular Pathophysiology [C] . Ashok K. Srivastava, Nihar R. Pandey, Antoine Blanc International Society for Heart Research.Congress . 2004

机译：血管平滑肌细胞氧化应激激活丝裂剂活化蛋白激酶和蛋白激酶B / Akt信号传导：血管病理生理学中的参与
5. The role of mitogen activated protein kinase, p44/42, in the butyltin-induced alterations of human natural killer cell function [D] . Dudimah, Fred D. 2010

机译：丝裂原活化蛋白激酶p44 / 42在丁基锡诱导的人类自然杀伤细胞功能改变中的作用
6. Role of the Protein Kinase C-ε–Raf-1–MEK-1/2–p44/42 MAPK Signaling Cascade in the Activation of Signal Transducers and Activators of Transcription 1 and 3 and Induction of Cyclooxygenase-2 After Ischemic Preconditioning [O] . Yu-Ting Xuan, Yiru Guo, Yanqing Zhu, -1

机译：蛋白激酶C-ε–Raf-1–MEK-1 / 2–p44 / 42 MAPK信号级联在缺血预处理后信号转导和转录1和3激活以及环加氧酶2诱导中的作用
7. Endothelial Nitric Oxide Synthase Plays an Obligatory Role in the Late Phase of Ischemic Preconditioning by Activating the Protein Kinase Cε–p44/42 Mitogen-Activated Protein Kinase–pSer-Signal Transducers and Activators of Transcription1/3 Pathway [O] . Yu-Ting Xuan, Yiru Guo, Yanqing Zhu, 2007

机译：通过激活蛋白激酶Cε-P44 / 42丝裂激活的蛋白激酶 - 信号传感器和转录1/3途径的活化剂，内皮一氧化氮合酶在缺血预处理的后期发挥了强制作用。

Endothelial Nitric Oxide Synthase Plays an Obligatory Role in the Late Phase of Ischemic Preconditioning by Activating the Protein Kinase C∊–p44/42 Mitogen-Activated Protein Kinase–pSer-Signal Transducers and Activators of Transcription1/3 Pathway

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