High Glucose Promotes Tumor Invasion and Increases Metastasis-Associated Protein Expression in Human Lung Epithelial Cells by Upregulating Heme Oxygenase-1 via Reactive Oxygen Species or the TGF-?21/PI3K/Akt Signaling Pathway

Xiaowen Kang; Fanwu Kong; Xiaomei Wu; Yeping Ren; Shijie Wu; Kaiming Wu; Zhe Jiang; Wei Zhang

首页> 外文期刊>Cellular Physiology and Biochemistry >High Glucose Promotes Tumor Invasion and Increases Metastasis-Associated Protein Expression in Human Lung Epithelial Cells by Upregulating Heme Oxygenase-1 via Reactive Oxygen Species or the TGF-?21/PI3K/Akt Signaling Pathway

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High Glucose Promotes Tumor Invasion and Increases Metastasis-Associated Protein Expression in Human Lung Epithelial Cells by Upregulating Heme Oxygenase-1 via Reactive Oxygen Species or the TGF-?21/PI3K/Akt Signaling Pathway

机译：高糖通过经由活性氧或TGF-β21/ PI3K / Akt信号通路上调血红素加氧酶-1来促进肿瘤侵袭并增加人肺上皮细胞转移相关蛋白的表达。

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《Cellular Physiology and Biochemistry》 |2015年第3期|共15页
作者
Xiaowen Kang; Fanwu Kong; Xiaomei Wu; Yeping Ren; Shijie Wu; Kaiming Wu; Zhe Jiang; Wei Zhang;
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中图分类细胞生物物理学;
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1. Reactive oxygen species and PI3K/Akt signaling play key roles in the induction of Nrf2-driven heme oxygenase-1 expression in sulforaphane-treated human mesothelioma MSTO-211H cells [J] . LeeY.-J., JeongH.-Y., KimY.-B., Food and Chemical Toxicology: An International Journal Published for the British Industrial Biological Research . 2012,第2期

机译：活性氧和PI3K / Akt信号传导在萝卜硫素治疗的人间皮瘤MSTO-211H细胞中诱导Nrf2驱动的血红素加氧酶-1表达中起关键作用
2. Angiotensin II-derived reactive oxygen species promote angiogenesis in human late endothelial progenitor cells through heme oxygenase-1 via ERK1/2 and AKT/PI3K pathways [J] . MaiJ., QiuQ., LinY.Q., Inflammation . 2014,第3期

机译：血管紧张素II衍生的活性氧通过ERK1 / 2和AKT / PI3K途径通过血红素加氧酶-1促进人晚期内皮祖细胞的血管生成
3. Transforming growth factor-beta1 stimulates heme oxygenase-1 expression via the PI3K/Akt and NF-kappaB pathways in human lung epithelial cells. [J] . Lin CC, Chiang LL, Lin CH, European Journal of Pharmacology: An International Journal . 2007,第2a3期

机译：转化生长因子-beta1通过人肺上皮细胞中的PI3K / Akt和NF-κB途径刺激血红素加氧酶-1的表达。
4. Increased trans-endothelial cells permeability induced by oxidized lipoprotein(a) relates to reactive oxygen species generation and desmogleins expression [C] . Zuo Wang, Jian Yang, Xiao-lei Zhang, World Congress on Medical Physics and Biomedical Engineering . 2013

机译：氧化脂蛋白（a）诱导的跨内皮细胞通透性增加与活性氧的产生和桥粒糖蛋白的表达有关
5. Crosstalk between the TGF-beta/Smad and PI3K/Akt pathways in the human epithelial MCF10A cell lineage and implications of signaling inhibitors. [D] . Zheng, Jie. 2009

机译：人上皮MCF10A细胞谱系中TGF-beta / Smad和PI3K / Akt通路之间的串扰和信号抑制剂的含义。
6. Nrf2-Heme Oxygenase-1 Attenuates High-Glucose-Induced Epithelial-to-Mesenchymal Transition of Renal Tubule Cells by Inhibiting ROS-Mediated PI3K/Akt/GSK-3β Signaling [O] . Jong Ho Shin, Kyeong Min Kim, Jin Uk Jeong, 2019

机译：Nrf2-血红素加氧酶-1通过抑制ROS介导的PI3K / Akt /GSK-3β信号减弱肾小管细胞的高糖诱导的上皮-间充质转化。
7. High Glucose Promotes Tumor Invasion and Increases Metastasis-Associated Protein Expression in Human Lung Epithelial Cells by Upregulating Heme Oxygenase-1 via Reactive Oxygen Species or the TGF-β1/PI3K/Akt Signaling Pathway [O] . Xiaowen Kang, Fanwu Kong, Xiaomei Wu, 2015

机译：高葡萄糖通过活性氧物种或TGF-β1/ pI3K / akt信号通路上调血红素加氧酶-1，促进肿瘤侵袭并增加人肺上皮细胞中转移相关蛋白的表达

High Glucose Promotes Tumor Invasion and Increases Metastasis-Associated Protein Expression in Human Lung Epithelial Cells by Upregulating Heme Oxygenase-1 via Reactive Oxygen Species or the TGF-?21/PI3K/Akt Signaling Pathway

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