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首页> 外文期刊>Case Reports in Obstetrics and Gynecology >A Case of Fetal Parvovirus B19 Myocarditis That Caused Terminal Heart Failure
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A Case of Fetal Parvovirus B19 Myocarditis That Caused Terminal Heart Failure

机译：导致末期心力衰竭的胎儿细小病毒B19心肌炎一例

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摘要

Parvovirus B19 is a well-established cause of fetal anemia and nonimmune fetal hydrops in pregnancy. Fetal parvovirus infection can cause severe destruction of erythroid progenitor cells, resulting in fetal anemia, hydrops, and intrauterine death. However, viral myocarditis with subsequent heart failure is another possible mechanism for hydrops formation as viral infection of fetal myocardial cells has been reported in postmortem examinations. We herein report a case of fetal cardiomegaly and massive pericardial effusion secondary to myocarditis as a result of parvovirus B19 infection. The case developed hydrops as consequence of severe anemia and experienced terminal heart failure, which led to the fetus dying an intrauterine death at 22 weeks of gestation. This case demonstrates that there may be an association between myocarditis caused by intrauterine parvovirus B19 infection and a poor outcome. The presence of viral myocarditis may be the determining prognostic factor in that situation.
机译：细小病毒B19是妊娠中胎儿贫血和非免疫性胎儿积水的公认原因。胎儿细小病毒感染可引起红细胞祖细胞的严重破坏，导致胎儿贫血，积水和子宫内死亡。然而，病毒性心肌炎伴随后的心力衰竭是积水形成的另一种可能的机制，因为在死后检查中已经报道了胎儿心肌细胞的病毒感染。我们在此报告了由于细小病毒B19感染导致继发于心肌炎的胎儿心脏肥大和大量心包积液。该病例因严重的贫血而发展为积液，并经历了晚期心力衰竭，这导致胎儿在妊娠22周时死于子宫内死亡。该病例表明，子宫内细小病毒B19感染引起的心肌炎与不良预后之间可能存在关联。病毒性心肌炎的存在可能是该情况下的决定性预后因素。

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