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首页> 外文期刊>Iranian red crescent medical journal >Pseudo-Foster Kennedy Syndrome as a Rare Presentation of Vitamin B12 Deficiency
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Pseudo-Foster Kennedy Syndrome as a Rare Presentation of Vitamin B12 Deficiency

机译：伪福斯特肯尼迪综合征为维生素B12缺乏症的罕见表现

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摘要

Pseudo-Foster Kennedy syndrome is a triad consisting of ipsilateral optic atrophy, contralateral optic disc edema, and ipsilateral anosmia in the absence of an intracranial mass. Vitamin B12 plays an important role in DNA synthesis, and its deficiency causes peripheral neuropathy, myeloneuropathy, and, very rarely, optic neuropathy. Case Presentation: In this study, we describe a 34-year-old male who presented with progressive loss of visual acuity and field. Fundoscopy showed optic disc edema with telangiectasia in the right eye, while the left eye had optic disc atrophy. We ruled out nearly all possible and common causes of optic neuropathy, and vitamin B12 deficiency was finally diagnosed. After treatment with vitamin B12, the patient improved. Conclusions: Demyelinating disease, anterior ischemic optic neuropathy, non-arteritic anterior ischemic optic neuropathy, autoimmune disease, and hereditary optic neuropathy could cause optic neuropathy. Normal CBC parameters and the absence of clinical manifestations of vitamin B12 deficiency could not rule out its diagnosis. Careful physical examinations and history-taking with a classical approach led us to the diagnosis of vitamin B12 deficiency and its treatment.
机译：假性福斯特肯尼迪综合征是由同侧视神经萎缩，对侧视盘水肿和无颅内肿物引起的同侧厌食症组成的三联征。维生素B12在DNA合成中起重要作用，维生素B12缺乏会引起周围神经病，脊髓神经病，很少会引起视神经病。病例介绍：在本研究中，我们描述了一名34岁男性，该男性表现为视力和视野逐渐丧失。眼底镜检查显示右眼视盘水肿伴毛细血管扩张，而左眼视盘萎缩。我们排除了几乎所有可能和常见的视神经病变原因，并最终诊断出维生素B12缺乏症。用维生素B12治疗后，患者情况有所好转。结论：脱髓鞘疾病，前部缺血性视神经病变，非动脉性前部缺血性视神经病变，自身免疫性疾病和遗传性视神经病变均可引起视神经病变。正常的CBC参数和缺乏维生素B12缺乏的临床表现不能排除其诊断。仔细的体格检查和采用经典方法的历史记录使我们得以诊断出维生素B12缺乏症及其治疗方法。

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