T cells and protective immunity to Plasmodium berghei in rats.

K N Brown; W Jarra; L A Hills

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T cells and protective immunity to Plasmodium berghei in rats.

机译：大鼠的T细胞和对伯氏疟原虫的保护性免疫。

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Experiments were carried out in which unfractionated spleen cells, and T lymphocyte subpopulations characterized by certain experimental criteria, were isolated at various times from rats infected with Plasmodium berghei. By adoptive transfer it was shown that unfractionated spleen cells, and T cells alone, could transfer protection to syngenic recipients as early as 11 days after infection of the cell donors. The protection conferred by T cells increased with the duration of the infection in the donors, at least up to 100 days. The additional presence of B cells in transferred lymphocyte populations enhanced their protective capacity over that shown by T cells alone. The role of T cells in protective immunity to malaria is discussed.

机译：进行实验，其中在不同时间从感染伯氏疟原虫的大鼠中分离出未分离的脾细胞和以某些实验标准为特征的T淋巴细胞亚群。通过过继转移显示，未分离的脾细胞和单独的T细胞最早可在感染细胞供体后11天将保护转移给同基因受体。 T细胞赋予的保护作用随着捐赠者感染时间的延长而增加，至少持续100天。与单独的T细胞相比，转移淋巴细胞群中B细胞的额外存在增强了它们的保护能力。讨论了T细胞在对疟疾的保护性免疫中的作用。

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《Infection and immunity》 |1976年第4期|共14页
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K N Brown; W Jarra; L A Hills;
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中图分类医学免疫学;
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1. γδ γδ T cells modulate humoral immunity against Plasmodium berghei Plasmodium berghei infection [J] . Inoue Shin‐Ichi, Niikura Mamoru, Asahi Hiroko, Immunology: An Official Journal of the British Society for Immunology . 2018,第4期

机译：γδγδT细胞调节体液免疫抗疟药疟原虫疟原虫感染
2. Gamma Interferon Production Is Critical for Protective Immunity to Infection with Blood-Stage Plasmodium berghei XAT but Neither NO Production nor NK Cell Activation Is Critical [J] . Toshihiko Yoneto, Takayuki Yoshimoto, Chrong-Reen Wang, Infection and immunity . 1999,第5期

机译：γ干扰素的产生对于保护伯氏疟原虫XAT感染的保护性免疫至关重要，但NO的产生和NK细胞的激活都不重要
3. Gamma Interferon Production Is Critical for Protective Immunity to Infection with Blood-Stage Plasmodium berghei XAT but Neither NO Production nor NK Cell Activation Is Critical [J] . Toshihiko Yoneto, Takayuki Yoshimoto, Chrong-Reen Wang, Infection and immunity . 1999,第5期

机译：γ干扰素的产生对于保护伯氏疟原虫XAT感染的保护性免疫至关重要，但NO的产生和NK细胞的激活都不重要
4. Combination of Pathogen-mimicking Polymer Particles and an Injectable, Synthetic Immune-Priming Center (sIPC) Significantly Enhances Cellular and Protective Immunity in Murine Models of Cancer [C] . Pallab Pradhan, Eileen Dawson, Jardin Leleux, Society for Biomaterials annual meeting and exposition . 2013

机译：模仿病原体的聚合物颗粒和可注射的合成免疫刺激中心（sIPC）的组合可显着增强癌症小鼠模型的细胞和保护性免疫力。
5. Studies on some aspects of pathogenesis of Plasmodium berghei infection in mice and rats. [D] . Green, George Andrew, IV. 1990

机译：在小鼠和大鼠中伯氏疟原虫感染的发病机理某些方面的研究。
6. T cells and protective immunity to Plasmodium berghei in rats. [O] . K N Brown, W Jarra, L A Hills 1976

机译：大鼠的T细胞和对伯氏疟原虫的保护性免疫。
7. Gamma Interferon Production Is Critical for Protective Immunity to Infection with Blood-Stage Plasmodium berghei XAT but Neither NO Production nor NK Cell Activation Is Critical [O] . Toshihiko Yoneto, Takayuki Yoshimoto, Chrong-Reen Wang, 1999

机译：Gamma干扰素的生产对于用血液阶段疟原虫感染的保护性豁免至关重要，但既不是生产和NK细胞激活都不至关重要
8. MHC Class I-Dependent Presentation of Exoerythrocytic Antigens to CD8+ TLymphocytes is Required for Protective Immunity against Plasmodium berghei [R] . White, K. L., Synder, H. L., Krzych, U. 1996

机译：对于CD8 + TL淋巴细胞，mHC I类依赖性的红细胞外抗原呈递是抗伯氏疟原虫的保护性免疫所必需的

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T cells and protective immunity to Plasmodium berghei in rats.

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