Peroxisome Proliferator-Activated Receptor γ and Retinoid X Receptor Agonists Have Minimal Effects on the Interaction of Endothelial Cells with Plasmodium falciparum- Infected Erythrocytes

Lena Serghides; Kevin C. Kain

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Peroxisome Proliferator-Activated Receptor γ and Retinoid X Receptor Agonists Have Minimal Effects on the Interaction of Endothelial Cells with Plasmodium falciparum- Infected Erythrocytes

机译：过氧化物酶体增殖物激活受体γ和类维生素A X受体激动剂对内皮细胞与恶性疟原虫感染的红细胞相互作用的影响最小

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Peroxisome proliferator-activated receptor γ-retinoid X receptor (PPARγ-RXR) agonists had minimal effects on the surface levels of CD36, intercellular cell adhesion molecule-1, or platelet-endothelial cell adhesion molecule-1 and had no effect on the cytoadherence of infected erythrocytes to either human umbilical vein endothelial cells or human microvascular endothelial cells or on malaria-induced interleukin-6 secretion from these cells. PPARγ-RXR agonists do not significantly modify malaria-infected erythrocyte-endothelial cell interactions in vitro.

机译：过氧化物酶体增殖物激活受体γ-类视黄醇X受体（PPARγ-RXR）激动剂对CD36，细胞间细胞粘附分子-1或血小板-内皮细胞粘附分子-1的表面水平影响最小，而对CD36的细胞粘附没有影响感染的红细胞进入人脐静脉内皮细胞或人微血管内皮细胞，或由疟疾诱导的这些细胞分泌白介素-6。 PPARγ-RXR激动剂在体外不能显着改变疟疾感染的红细胞-内皮细胞的相互作用。

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《Infection and immunity》 |2005年第2期|共5页
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Lena Serghides; Kevin C. Kain;
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中图分类医学免疫学;
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1. Peroxisome proliferator-activated receptor gamma-retinoid X receptor agonists increase CD36-dependent phagocytosis of Plasmodium falciparum-parasitized erythrocytes and decrease malaria-induced TNF-alpha secretion by monocytes/macrophages. [J] . Serghides L, Kain KC The Journal of Immunology: Official Journal of the American Association of Immunologists . 2001,第11期

机译：过氧化物酶体增殖物激活的受体γ-类维生素A X受体激动剂可增加恶性疟原虫寄生的红细胞的CD36依赖性吞噬作用，并减少疟疾诱导的单核细胞/巨噬细胞分泌的TNF-α。
2. Peroxisome Proliferator-Activated Receptor γ-Retinoid X Receptor Agonists Increase CD36-Dependent Phagocytosis of Plasmodium falciparum-Parasitized Erythrocytes and Decrease Malaria-Induced TNF-α Secretion by Monocytes/Macrophages [J] . Lena Serghides, Kevin C. Kain The journal of immunology . 2001,第11期

机译：过氧化物酶体增殖物激活的受体γ-类维生素A X受体激动剂可增加恶性疟原虫寄生的红细胞的CD36依赖性吞噬作用，并减少单核细胞/巨噬细胞引起的疟疾诱导的TNF-α分泌。
3. Peroxisome proliferator-activated receptor-gamma and retinoid X receptor agonists synergistically suppress proliferation of immortalized endometrial stromal cells. [J] . Wu Y, Guo SW Fertility and Sterility: Official Journal of the American Fertility Society, Pacific Coast Fertility Society, and the Canadian Fertility and Andrology Society . 2009,第5Suppla期

机译：过氧化物酶体增殖物激活受体-γ和类维生素A X受体激动剂可协同抑制永生化子宫内膜基质细胞的增殖。
4. α-Naphtyl Substituted Pirinixic Acid Derivatives as Potent Selective Agonists of the Peroxisome Proliferator-Activated Receptor Gamma [C] . Theresa M. Thieme, Ramona Steri, Ewgenij Proschak European Symposium on Organic Chemistry . 2009

机译：α-萘基取代的吡啶酸衍生物作为过氧化物体增殖物激活的受体γ的有效选择性激动剂
5. Peroxisome Proliferator-activated Receptor-γ (PPARγ) Agonists: Potential Novel Treatment for HIV-1 Associated Brain Inflammation [D] . Omeragic, Amila . 2020

机译：过氧化物体增殖物激活受体-γ（PPARγ）激动剂：对HIV-1相关脑炎症的潜在新待遇
6. Peroxisome Proliferator-Activated Receptor γ and Retinoid X Receptor Agonists Have Minimal Effects on the Interaction of Endothelial Cells with Plasmodium falciparum- Infected Erythrocytes [O] . Lena Serghides, Kevin C. Kain 2005

机译：过氧化物酶体增殖物激活的受体γ和类维生素A X受体激动剂对内皮细胞与恶性疟原虫感染的红细胞相互作用的影响最小
7. Peroxisome Proliferator-Activated Receptor γ and Retinoid X Receptor Agonists Have Minimal Effects on the Interaction of Endothelial Cells with Plasmodium falciparum- Infected Erythrocytes [O] . Serghides, Lena, Kain, Kevin C. 2005

机译：过氧化物酶体增殖物激活的受体γ和类维生素A X受体激动剂对内皮细胞与恶性疟原虫感染的红细胞相互作用的影响最小

Peroxisome Proliferator-Activated Receptor γ and Retinoid X Receptor Agonists Have Minimal Effects on the Interaction of Endothelial Cells with Plasmodium falciparum- Infected Erythrocytes

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