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首页> 外文期刊>Journal of International Medical Research >Budd–Chiari syndrome as a complication of eosinophilic granulomatosis with polyangiitis in a young Chinese man: a case report
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Budd–Chiari syndrome as a complication of eosinophilic granulomatosis with polyangiitis in a young Chinese man: a case report

机译：Budd-Chiari综合征作为嗜酸性粒细胞粒细胞瘤的复杂性与年轻人的嗜酸性粒细胞瘤，案例报告

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Hepatic vein thrombosis is a rare occurrence in the clinical course of eosinophilic granulomatosis with polyangiitis (EGPA). The major mechanism of thrombosis has been postulated to involve the release of toxic proteins from eosinophils. A 36-year-old man with EGPA was admitted to our hospital in July 2018 with hematemesis and melena. Findings on physical examination included ascites and pigmentation of the lower extremities. Ultrasonography of the hepatic vein and inferior vena cava showed an obstruction of the hepatic vein. Magnetic resonance imaging showed low enhancement in the right hepatic vein region. At 34 years of age, the patient’s EGPA had initially presented as asthma with eosinophilia (white blood cell count of 11.46?×?10 ~(12)/L with 14.6% eosinophils). His skin biopsy showed infiltration of inflammatory cells and eosinophils, especially around medium-sized vessels, which was consistent with EGPA. The patient was thus diagnosed with Budd–Chiari syndrome associated with EGPA.
机译：肝静脉血栓形成是嗜酸性粒细胞粒细胞瘤的临床过程中的罕见发生（EGPA）。血栓形成的主要机制已经假设涉及从嗜酸性粒细胞释放有毒蛋白质。 2018年7月，一名带有EGPA的36岁的男子们在2018年7月录取了我们的医院，占据了呕血和Melena。物理检查的结果包括下肢的腹水和色素沉着。肝静脉和下腔静脉的超声检查显示肝静脉的阻塞。磁共振成像显示出右肝静脉区域的低增强。在34岁时，患者的EGPA最初呈现为嗜酸性粒细胞的哮喘（白细胞计数为11.46Ω·×10〜（12）/ L，具有14.6％的嗜酸性粒细胞）。他的皮肤活组织检查显示出炎症细胞和嗜酸性粒细胞的浸润，尤其是围绕中型容器，其与EGPA一致。因此，患者被诊断出与EGPA相关的Budd-Chiari综合征。

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