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机译:空腹诱导的蛋白磷酸酶1调节亚基有助于通过调节肝糖原的餐后血糖稳态。
From the Key Laboratory of Nutrition and Metabolism, Institute for Nutritional Sciences, Shanghai Institutes for Biological Sciences, Graduate School of the Chinese Academy of Sciences, Shanghai, ChinaCorresponding author: Yan Chen, ychen3@sibs.ac.cn.Received 30 November 2010 and accepted 25 February 2011.DOI: 10.2337/db10-1663This article contains Supplementary Data online at http://diabetes. diabetesjournals.org/lookup/suppl/doi:10.2337/dbl0-1663/-/DCl.© 2011 by the American Diabetes Association. Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creativecommons.org/licenses/by -nc-nd/3.0/ for details.;
机译:空腹诱导的蛋白磷酸酶1调节亚基有助于通过调节肝糖原的餐后血糖稳态。
机译:肝蛋白磷酸酶1调节亚基3B(Ppp1r3b)促进肝糖原合成,从而调节空腹能量稳态
机译:肝蛋白磷酸酶1调节亚基3B(Ppp1r3b)促进肝糖原合成,从而调节空腹能量稳态
机译:使用定量质谱法在大鼠β-胰岛细胞中蛋白质磷酸酶2A催化亚基的蛋白质相互作用伴侣
机译:小鼠长期蛋白质磷酸酶2A抑制作用是否会异常调节外周葡萄糖稳态,降低肝糖原含量并损害肝脏中胰岛素介导的信号转导?
机译:空腹诱导的蛋白磷酸酶1调节亚基有助于通过调节肝糖原的餐后血糖稳态。
机译:空腹诱导的蛋白磷酸酶1调节亚基有助于通过调节肝糖原的餐后血糖稳态。