A novel 3'-end repair mechanism in an RNA virus

Peter D. Nagy; Clifford D. Carpenter; Anne E. Simon

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A novel 3'-end repair mechanism in an RNA virus

机译：RNA病毒中的新型3'末端修复机制

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Many positive-stranded RNA viruses con- tain short, single-stranded 3' ends that are vulnerable to degradation by host cellular RNases. Therefore, the existence of a 3'-end repair mechanism (analogous to cellular telomer- ases) must be required and/or advantageous for RNA viruses. Accordingly, we provide evidence suggesting that deletions of up to 6 nt from the 3' end of satellite (sat-) RNA C (a small parasitic RNA associated with turnip crinkle carmovirus) are repaired to the wild-type sequence in vivo and in vitro.

机译：许多正链RNA病毒具有短的单链3'末端，容易被宿主细胞的RNase降解。因此，对于RNA病毒，必须存在3'端修复机制（类似于细胞端粒）和/或有利于RNA病毒。因此，我们提供的证据表明，在体内和体外，卫星（sat）RNA C（与芜菁皱纹细小病毒相关的小寄生RNA）的3'末端最多缺失了6 nt的缺失被修复为野生型序列。

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《Proceedings of the National Academy of Sciences of the United States of America》 |1997年第4期|p.1113-1118|共6页
作者
Peter D. Nagy; Clifford D. Carpenter; Anne E. Simon;
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收录信息美国《科学引文索引》(SCI);美国《生物学医学文摘》(MEDLINE);美国《化学文摘》(CA);
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正文语种 eng
中图分类自然科学总论;
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A novel 3'-end repair mechanism in an RNA virus

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