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机译:Aurora B激酶磷酸化并促使p53降解
Graduate School of Biomedical Sciences, University of Texas, Houston, TX 77030,The M. D. Anderson Cancer Center, Program in Genes and Development,University of Texas, Houston, TX 77330,Department of Molecular and Cellular Oncology, The M. D. Anderson Cancer Center, University of Texas, Houston, TX 77030;
Graduate School of Biomedical Sciences, University of Texas, Houston, TX 77030,Department of Molecular and Cellular Oncology, The M. D. Anderson Cancer Center, University of Texas, Houston, TX 77030,The M. D. Anderson Cancer Center, Program in Cancer Biology, University of Texas, Houston, TX 77330;
Graduate School of Biomedical Sciences, University of Texas, Houston, TX 77030,Department of Molecular and Cellular Oncology, The M. D. Anderson Cancer Center, University of Texas, Houston, TX 77030,The M. D. Anderson Cancer Center, Program in Cancer Biology, University of Texas, Houston, TX 77330;
Graduate School of Biomedical Sciences, University of Texas, Houston, TX 77030,Department of Molecular and Cellular Oncology, The M. D. Anderson Cancer Center, University of Texas, Houston, TX 77030;
Graduate School of Biomedical Sciences, University of Texas, Houston, TX 77030,Department of Molecular and Cellular Oncology, The M. D. Anderson Cancer Center, University of Texas, Houston, TX 77030,The M. D. Anderson Cancer Center, Program in Cancer Biology, University of Texas, Houston, TX 77330;
Graduate School of Biomedical Sciences, University of Texas, Houston, TX 77030,The M. D. Anderson Cancer Center, Program in Genes and Development,University of Texas, Houston, TX 77330,Department of Molecular and Cellular Oncology, The M. D. Anderson Cancer Center, University of Texas, Houston, TX 77030;
Department of Molecular and Cellular Oncology, The M. D. Anderson Cancer Center, University of Texas, Houston, TX 77030;
Department of Molecular and Cellular Oncology, The M. D. Anderson Cancer Center, University of Texas, Houston, TX 77030;
Department of Genetics, The M. D. Anderson Cancer Center, University of Texas, Houston, TX 77030;
Graduate School of Biomedical Sciences, University of Texas, Houston, TX 77030,Department of Molecular and Cellular Oncology, The M. D. Anderson Cancer Center, University of Texas, Houston, TX 77030,The M. D. Anderson Cancer Center, Program in Cancer Biology, University of Texas, Houston, TX 77330;
Graduate School of Biomedical Sciences, University of Texas, Houston, TX 77030,Department of Molecular and Cellular Oncology, The M. D. Anderson Cancer Center, University of Texas, Houston, TX 77030,The M. D. Anderson Cancer Center, Program in Cancer Biology, University of Texas, Houston, TX 77330;
Graduate School of Biomedical Sciences, University of Texas, Houston, TX 77030,Department of Molecular and Cellular Oncology, The M. D. Anderson Cancer Center, University of Texas, Houston, TX 77030;
Graduate School of Biomedical Sciences, University of Texas, Houston, TX 77030,Department of Molecular and Cellular Oncology, The M. D. Anderson Cancer Center, University of Texas, Houston, TX 77030,The M. D. Anderson Cancer Center, Program in Cancer Biology, University of Texas, Houston, TX 77330;
Graduate School of Biomedical Sciences, University of Texas, Houston, TX 77030,The M. D. Anderson Cancer Center, Program in Genes and Development,University of Texas, Houston, TX 77330,Department of Molecular and Cellular Oncology, The M. D. Anderson Cancer Center, University of Texas, Houston, TX 77030;
Graduate School of Biomedical Sciences, University of Texas, Houston, TX 77030,Department of Molecular and Cellular Oncology, The M. D. Anderson Cancer Center, University of Texas, Houston, TX 77030;
Department of Molecular and Cellular Oncology, The M. D. Anderson Cancer Center, University of Texas, Houston, TX 77030;
Department of Molecular and Cellular Oncology, The M. D. Anderson Cancer Center, University of Texas, Houston, TX 77030;
Graduate School of Biomedical Sciences, University of Texas, Houston, TX 77030,Department ofEmergency Medicine, The M. D. Anderson Cancer Center, University of Texas, Houston,TX 77030,Department of Endocrine Neoplasia and Hormonal Disorders, The M. D. Anderson Cancer Center, University of Texas, Houston, TX 77030;
Graduate School of Biomedical Sciences, University of Texas, Houston, TX 77030,The M. D. Anderson Cancer Center, Program in Genes and Development,University of Texas, Houston, TX 77330,Department of Molecular and Cellular Oncology, The M. D. Anderson Cancer Center, University of Texas, Houston, TX 77030,The M. D. Anderson Cancer Center, Program in Cancer Biology, University of Texas, Houston, TX 77330;
机译:通过使用siRNA或选择性Aurora激酶抑制剂PHA680632抑制p53缺陷癌细胞中的Aurora-A激酶来增强放射反应
机译:两种裂解沟激酶Aurora-B和Rho激酶/ ROCK独立调节胞质分裂的进程:可能存在新的裂解沟激酶使ezrin / radixin / moesin(ERM)磷酸化。
机译:卵裂沟中结节中磷酸化的特定位点的功能意义:Aurora-B在胞质分裂过程中可能与Rho激酶协同磷酸化并调节III型中间丝
机译:MAP4K4是磷酸化Farp1的苏氨酸激酶
机译:与Mdm2复合的丝氨酸15磷酸化p53被抑制激活p53效应子途径。
机译:PNAS Plus:Aurora B激酶磷酸化并促使p53降解
机译:Aurora B激酶磷酸化并促使p53降解
机译:sV40大肿瘤抗原和肿瘤抑制因子p53被来自人细胞的DNa活化蛋白激酶磷酸化。