机译:αβTCR功能方面的证据
Department of Microbiology and Immunology, Chevy Chase, MD 20815 Stanford University School of Medicine, Stanford, CA 94305;
rnDepartment of Microbiology and Immunology, Chevy Chase, MD 20815 Stanford University School of Medicine, Stanford, CA 94305 Graduate Program in Immunology, Chevy Chase, MD 20815;
rnStanford University School of Medicine, Stanford, CA 94305 Graduate Program in Biophysics, Chevy Chase, MD 20815;
rnStanford University School of Medicine, Stanford, CA 94305 CCIS/ITI Summer High School Research Program, Chevy Chase, MD 20815;
rnDepartment of Microbiology and Immunology, Chevy Chase, MD 20815 Stanford University School of Medicine, Stanford, CA 94305;
rnDepartment of Microbiology and Immunology, Chevy Chase, MD 20815 Stanford University School of Medicine, Stanford, CA 94305;
rnDepartment of Microbiology and Immunology, Chevy Chase, MD 20815 Stanford University School of Medicine, Stanford, CA 94305;
rnDepartment of Microbiology and Immunology, Chevy Chase, MD 20815 Stanford University School of Medicine, Stanford, CA 94305 Nomis Center for Immunobiology and Microbial Pathogenesis, Waitt Advanced Biophotonics Center, the Salk Institute, La Jolla, CA 92037;
rnDepartment of Microbiology and Immunology, Chevy Chase, MD 20815 Stanford University School of Medicine, Stanford, CA 94305 Immunology Program, Memorial Sloan-Kettering Cancer Center, New York, NY 10021;
rnDepartment of Microbiology and Immunology, Chevy Chase, MD 20815 Stanford University School of Medicine, Stanford, CA 94305;
rnStanford University School of Medicine, Stanford, CA 94305 Department of Molecular and Cellular Physiology, Chevy Chase, MD 20815 Department of Structural Biology, Chevy Chase, MD 20815 The Howard Hughes Medical Institute, Chevy Chase, MD 20815;
rnDepartment of Microbiology and Immunology, Chevy Chase, MD 20815 Stanford University School of Medicine, Stanford, CA 94305 The Howard Hughes Medical Institute, Chevy Chase, MD 20815;
T cell; T cell receptor; CD3; organization; dimerization; signaling;
机译:pTα缺陷小鼠的等位基因排斥:没有证据表明两个T细胞受体(TCR)-β链的细胞表面表达,但在功能性TCR-β转基因存在下抑制内源性Vβ→(D)Jβ重排的效率较低
机译:1-丙基-1-亚硝基脲诱导的胸腺淋巴瘤中TCR-β,TCR-γ,CD4和CD8的过度表达:破坏TCR-β的等位基因排斥并在淋巴瘤cFTL53上表达功能性TCR-βγ异二聚体。
机译:保守的TCR CDR#alpha#3循环对接的功能证据控制了密切相关肽的Cross0识别:I类复合物。
机译:Postcrisis时代的分析Ontransformational Readership:GE中来自杰克韦尔奇的证据
机译:盖层岩性岩性的岩石强度:过去的岩性破坏,流体运移以及露头和地下储层岩性界面分析的证据。
机译:αβTCR功能方面的证据
机译:αβTCR功能方面的证据