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机译:NOD Idd3基因座中IL-2和IL-21之间的奇偶性丧失
Department of Immunology, Garvan Institute of Medical Research, 384 Victoria Street, Darlinghurst NSW 2010, Australia University of New South Wales, School of Biotechnology and Biomolecular Sciences, UNSW, Sydney 2052, Australia;
Department of Immunology, Garvan Institute of Medical Research, 384 Victoria Street, Darlinghurst NSW 2010, Australia;
Centre for Diabetes and Metabolic Medicine, Institute of Cell and Molecular Sciences, Barts/Queen Mary's School of Medicine and Dentistry, 4 Newark Street, London E1 2AT, United Kingdom School of Life Sciences, Kingston University, Surrey KT1 1LQ, United Kingdom;
Center for Infectious Medicine, Department of Medicine, Karolinska Institutet, Karolinska University Hospital Huddinge, F59, S-141 86 Stockholm, Sweden;
Department of Immunology, Garvan Institute of Medical Research, 384 Victoria Street, Darlinghurst NSW 2010, Australia St. Vincent's Clinical School, University of NSW, Sydney, NSW, Sydney, NSW, Australia and WCU-IBB Program, POSTECH, Korea St. Vincent's Clinical School, University of NSW, Sydney, NSW 2010, Australia;
Department of Immunology, Garvan Institute of Medical Research, 384 Victoria Street, Darlinghurst NSW 2010, Australia St. Vincent's Clinical School, University of NSW, Sydney, NSW 2010, Australia;
type 1 diabetes; cytokines; transcription factor; allele; promoter;
机译:NOD小鼠中CTLA-4的缺陷诱导受Idd3 / IL-2的NOD等位基因和染色体1上的新型基因座(Ctex)端粒控制。
机译:最小启动子上游的SNP控制IL-2表达,并且是自身免疫疾病易感性基因座Aod2 / Idd3 / Eae3的候选者。
机译:无法解释的不育症患者的血清IL-2,IL-4,IL-6,IL-8,IL-21,TNFα,IFNγ和Tfh / CD4 T细胞比率增加:Tfh和CD-21的增加与存在密切相关自身抗体
机译:基因座:用于测量火山羽毛的多机器人丢失算法
机译:经过工程改造以过度表达IL-2,IL-7,IL-15或IL-21的肿瘤特异性人类原代T细胞可在免疫缺陷小鼠模型中赋予不同程度的肿瘤排斥反应和异种移植物抗宿主病。
机译:NOD Idd3基因座中IL-2和IL-21之间的奇偶性丧失
机译:NOD Idd3基因座中IL-2和IL-21之间的奇偶性丧失