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首页> 外文期刊>Physiological Zoology >Effects of hypervolemia and hypovolemia on cardiac and posterior lymph heart function in the toad Bufo marinus (L.)
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Effects of hypervolemia and hypovolemia on cardiac and posterior lymph heart function in the toad Bufo marinus (L.)

机译：高血容量和低血容量对蟾蜍蟾蜍心和后淋巴心功能的影响

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Cardiac and posterior lymph heart function in the toad Bufo marinus (L.) were measured in response to hemorrhage and both systemic and lymphatic volume loading to evaluate the role of the amphibian lymphatic system in fluid regulation. In every animal, systemic volume loading elicited an initial sharp rise in mean arterial blood pressure, corresponding to an immediate cessation of lymph heart contraction lasting approximately 12 min. This was followed by a short-term decrease in blood pressure, to 36 percent below normal. While stroke volume and lymph flow for the individual animals increased variably over a 6-h period, mean lymph heart rate and pressure varied little. Lymphatic volume loading elicited only minor changes in blood heart and lymph heart rate. Again, stroke volume and lymph flow increased variably, with the effect lasting about 13 h. Hypovolemia caused a sharp drop in blood pressure and a corresponding rise in blood heart rate. Lymph heart variables changed little, with the exception of rate, which, in some animals, was elevated for approximately 20 min following hemorrhage.
机译：蟾蜍蟾蜍（L.）蟾蜍的心脏和后淋巴心脏功能通过对出血以及全身和淋巴体积负荷的反应进行测量，以评估两栖淋巴系统在体液调节中的作用。在每只动物中，全身体积负荷引起平均动脉血压的初始急剧上升，这对应于持续约12分钟的立即停止的淋巴心脏收缩。随后血压短期下降，比正常低36％。尽管每只动物的中风量和淋巴流量在6小时内都有不同程度的增加，但平均淋巴心率和压力变化不大。淋巴体积负荷仅引起血液心脏和淋巴心率的微小变化。再次，中风量和淋巴流量增加，持续约13小时。低血容量导致血压急剧下降，相应地使血液心率上升。淋巴的心脏变量几乎没有变化，除了速率，在某些动物中，出血后升高了约20分钟。

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