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首页> 外文期刊>Progress in brain research >Anosognosia for left-sided motor and sensory deficits, motor neglect, and sensory hemiinattention: is there a relationship?
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Anosognosia for left-sided motor and sensory deficits, motor neglect, and sensory hemiinattention: is there a relationship?

机译：左侧运动和感觉缺陷，运动疏忽和感觉偏执的厌食症：有关系吗？

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In recent years, research on unilateral spatial neglect has focused on dissociations between different aspects of the clinical syndrome, which is now considered by many students as a multi-componential disorder. Notwithstanding this leading view, there is at least one empirical argument which supports a unitary interpretation of the disorder. This is based on the observation, now replicated many times, that a variety of sensory stimulations (vestibular, optokinetic, transcutaneous mechanical vibration and nervous electrical, visual prism adaptation) involving a lateral change (left-right asymmetry) in the input pattern, affect in a very similar fashion virtually all manifestations of the syndrome, including: visuo-spatial neglect; hemianaesthesia (somatosensory hemi-inattention); extinction, hemiparesis, hemiplegia, and anosognosia for these motor disorders; somatoparaphrenia. These effects may be accounted for with reference to a spatial medium, articulated in a number of specific components, which ismodulated by sensory input in a fundamentally similar fashion. Recent investigations concerning the neural bases of some of these stimulations support this view. In this chapter the case of the co-variation of the effects of vestibular stimulation on motor deficits and on anosognosia for hemiplegia is considered. The suggestion is made that one mechanism underlying anosognosia for hemiplegia is unawareness of a deficit of intention, or movement planning component, rather than, or in addition to, unawareness of a primary motor deficit. Temporary remission of anosognosia after vestibular stimulation may represent recovery from this neglect-related component, of which, as of other manifestations of the syndrome, patients are typically unaware. The recovered intention to move may allow the detection by the patient of the presence of a residual primary motor deficit, through a feedback mechanism.
机译：近年来，对单方面空间忽视的研究集中于临床综合症不同方面之间的分离，该现象现已被许多学生视为多成分疾病。尽管有这种主导观点，但至少有一个经验论证支持对疾病的统一解释。这是基于观察到的结果，现在已经重复了很多次，观察到输入模式中存在横向变化（左右不对称）的各种感觉刺激（前庭，视动，经皮机械振动以及神经电，视觉棱镜适应）会影响该综合征的几乎所有表现都非常相似，包括：视觉空间疏忽;呼吸麻痹（体感半不全）;这些运动障碍的绝种，偏瘫，偏瘫和失语症;躯体旁肾炎。这些影响可以参考以许多特定部件铰接的空间介质来解释，该空间介质以基本上相似的方式由感觉输入来调节。关于这些刺激中的一些神经基础的最新研究支持该观点。在本章中，考虑前庭刺激对运动功能障碍和偏瘫的失语症的协变量。有人提出，偏瘫的失语症的一种潜在机制是对意识缺乏或运动计划成分的不了解，而不是对原发性运动缺乏的了解，或除此以外还没有意识到。前庭刺激后暂时的失音症缓解可能表示已从这一与疏忽有关的成分中恢复过来，与该综合征的其他表现一样，患者通常没有意识到。恢复的移动意图可以允许患者通过反馈机制检测到残留的主要运动功能障碍。

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