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首页> 外文期刊>The Bovine Practitioner >The Magnitude and Patterns of Ruminai Hydrogen Sulfide Production, Blood Thiamine Concentration, and Mean Pulmonary Arterial Pressure in Feedlot Steers Consuming Water of Different Sulfate Concentrations
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The Magnitude and Patterns of Ruminai Hydrogen Sulfide Production, Blood Thiamine Concentration, and Mean Pulmonary Arterial Pressure in Feedlot Steers Consuming Water of Different Sulfate Concentrations

机译：饲喂不同硫酸盐浓度的水的育肥场中反刍动物产生的硫化氢，血硫胺素浓度和平均肺动脉压的大小和模式

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Twenty-seven single-source, yearling crossbred steers (713 Ib; 324 kg) were used to evaluate the effect of water sulfate concentration on the patterns and magnitude of ruminai gas cap hydrogen sulfide, blood thia-mine concentration and mean pulmonaryarterial blood pressure (mPAP). Target water sulfate concentrations were 125, 500 and 2000 mg sulfate/L of drinking water, while the observed water sulfate concentrations were 136, 583 and 2,360 mg/L. Ruminai gas cap hydrogen sulfide concentrations weremeasured three times per week for 10 weeks, then once a week for seven additional weeks. On days 29 and 113, whole-blood samples were collected and analyzed for thiamine concentration. Pulmonary arterial blood pressures were measured on day 100. Ruminaigas cap hydrogen sulfide concentrations decreased to day 15, then increased and peaked by day 31, and finally decreased and remained relatively stable. The peak in hydrogen sulfide concentration was associated with an event of polioencephalomalacia in the treatment group consuming water with 2,360 mg/L sulfate, and the greatest frequency of polioencephalomalacia events in cattle in an adjacent commercial feedlot consuming water with a similar sulfate concentration. Ruminai hydrogen sulfide concentrationincreased with increasing water sulfate concentration (P < 0.05). A treatment effect on blood thiamine concentration was not detected (P = 0.29). Mean pulmonary arterial pressure increased linearly with increasing water sulfate concentration (P < 0.05),Ruminai gas cap hydrogen sulfide concentrations greater than 1000 ppm provide evidence of excessive sulfur consumption. Pathological concentrations of ruminai sulfides appear central to the pathogenesis of sulfur-associated polioencephalomalacia.
机译：用二十七只单源一岁杂交杂交ers牛（713 Ib; 324 kg）评估硫酸水浓度对瘤胃气帽硫化氢，血硫胺浓度和平均肺动脉血压的模式和大小的影响（ mPAP）。饮用水中的目标硫酸盐浓度为125、500和2000 mg硫酸/ L，而观察到的硫酸盐水浓度为136、583和2360 mg / L。每周测量三次Ruminai气帽硫化氢浓度，持续10周，然后每周测量一次，持续7周。在第29和113天，收集全血样品并分析硫胺素浓度。在第100天测量肺动脉血压。Ruminaigas上限的硫化氢浓度降低至第15天，然后在第31天升高并达到峰值，最后降低并保持相对稳定。硫化氢浓度的峰值与在处理组中使用硫酸盐浓度为2,360 mg / L的治疗组发生脊髓灰质软化病的事件有关，并且在相邻的商业饲养场中，硫酸盐浓度与之相似的饮用水中，脊髓灰质软化病的发生频率最高。 Ruminai硫化氢浓度随硫酸盐水浓度的增加而增加（P <0.05）。未检测到对血硫胺素浓度的治疗效果（P = 0.29）。平均肺动脉压随硫酸水浓度的增加而线性增加（P <0.05），Ruminai气顶硫化氢浓度大于1000 ppm则提供了过量硫消耗的证据。瘤胃硫化物的病理学浓度似乎是与硫有关的脊髓灰质脑瘤的发病机理的中心。

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