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首页> 外文期刊>Protoplasma: An International Journal of Cell Biology >Phloem loading in the sucrose-export-defective (SXD-1) mutant maize is limited by callose deposition at plasmodesmata in bundle sheath-vascular parenchyma interface
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Phloem loading in the sucrose-export-defective (SXD-1) mutant maize is limited by callose deposition at plasmodesmata in bundle sheath-vascular parenchyma interface

机译：蔗糖出口缺陷型（SXD-1）突变型玉米中的韧皮部负载受束鞘在血管鞘实质薄壁界面中的浆膜下的call沉积限制

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Using Lucifer Yellow we have demonstrated that the phloem-loading pathway from the mesophyll to the bundle sheath-vascular parenchyma interface in Zea mays source leaves follows a symplasmic route in small and intermediate vascular bundles in control as well as in the green sections of mutant sucrose-export-defective (SXD-1) plants. In the anthocyanin-rich mutant leaf sections, Lucifer Yellow transport was prohibited along the same path, at the bundle sheath-vascular parenchyma interface in particular. Plasmodesmata at the latter interface in SXD-1 anthocyanin-rich leaf sections appear to be structurally altered through callose deposition at the plasmodesmal orifices. We suggest that a transport bottleneck at the bundle sheath-vascular parenchyma interface is thus orchestrated and regulated through callose formation, preventing symplasmic transport across this important loading interface. [References: 34]
机译：使用路西法黄，我们已经证明了玉米叶中从叶肉到束鞘-血管薄壁组织界面的韧皮部加载途径在对照和突变蔗糖的绿色部分中的中小血管束中均遵循同质途径-出口缺陷（SXD-1）植物。在富含花青素的突变叶片切片中，特别是在束鞘-血管薄壁组织界面处，禁止沿相同路径进行荧光素黄运输。 SXD-1富含花青素的叶部分中后一个界面处的等离子线虫似乎通过在等离子孔处的ose质沉积而在结构上发生了变化。我们建议通过sheath质的形成来协调和调节束鞘-血管薄壁组织界面处的运输瓶颈，从而防止通过该重要负荷界面的同质运输。 [参考：34]

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