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首页> 外文期刊>The Veterinary Journal >Studies on growth cartilage in the horse and their application toaetiopathogenesis of dyschondroplasia (osteochondrosis) [Review]
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Studies on growth cartilage in the horse and their application toaetiopathogenesis of dyschondroplasia (osteochondrosis) [Review]

机译：马生长软骨及其在软骨发育不良（骨软骨病）发病机理中的应用研究[综述]

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摘要

The importance of osteochondrosis (dyschondroplasia) to the hot-se industry has been well documented since it was first recorded 50 years ago. The condition is known to be multifactorial in origin, arising from focal failure of endochondral ossification at predilection sites in articular/epiphyseal growth cartilage, but specific information on its aetiopathogenesis is sparse. This paper reviews the current knowledge of growth cartilage metabolism and the process of normal endochondral ossification in the horse. It highlights the localization of various protein products of chondrocytes and the differences in the zones of articular cartilage. In the early focal lesions (referred to as dyschondroplasia) there are alterations in the chondrocytes, extracellular matrix and some of the local protein products. The most obvious feature is an alteration in matrix metabolism which may be responsible fur triggering a range of other factors leading to the development of a retained core of cartilage and a primary lesion of dyschondroplasia.
机译：自从50年前首次记录以来，骨软骨病（软骨发育不良）对热门行业的重要性已得到充分证明。已知该病的起源是多因素的，这是由于关节/上phy软骨生长的好发部位软骨内骨化的局灶性衰竭引起的，但是有关其发病机理的具体信息很少。本文回顾了目前有关马软骨生长代谢和正常软骨内骨化过程的知识。它强调了软骨细胞各种蛋白质产物的定位以及关节软骨区域的差异。在早期局灶性病变（称为软骨发育不良）中，软骨细胞，细胞外基质和某些局部蛋白质产物发生改变。最明显的特征是基质代谢的改变，这可能是引发一系列其他因素的原因，这些因素导致保留的软骨核心和软骨发育不良的原发灶的发展。

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