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首页> 外文期刊>Zoological Science >Effects of lethal levels of environmental hypercapnia on cardiovascular and blood-gas status in yellowtail, Seriola quinqueradiata
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Effects of lethal levels of environmental hypercapnia on cardiovascular and blood-gas status in yellowtail, Seriola quinqueradiata

机译：致命的环境高碳酸血症水平对五尾tail（Seriola quinqueradiata）的心血管和血气状态的影响

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The cardiorespiratory responses were examined in yellowtail, Seriola quinqueradiata exposed to two levels of hypercapnia. (seawater equilibrated with a gas mixture containing 1% CO2 (water PCO2 = 7 mmHg) or 5% CO2 (38 mmHg)) for 72 hr at 20degreesC. Mortality was 100% within 8 hr at 5% CO2, while no fish died at 1% CO2. No cardiovascular variables (cardiac output, (Q) over dot; heart rate, HR; stroke volume, SV and arterial blood pressure, BP) significantly changed from pre-exposure values during exposure to 1% CO2. Arterial CO2 partial pressure (PaCO2) Significantly increased (P < 0.05), reaching a new steady-state level after 3 hr. Arterial blood pH (pHa) decreased initially (P < 0.05), but was subsequently restored by elevation of plasma bicarbonate ([HCO3-]). Arterial O-2 partial pressure (PaO2), oxygen content (CaO2), and hematocrit (Hct) were maintained throughout the exposure period. In contrast, exposure to 5% CO2 dramatically reduced (Q) over dot (P < 0.05) through decreasing SV (P < 0.05), although HR did not change. BP was transiently elevated (P < 0.05), followed by a precipitous fall before death. The pHa was restored incompletely despite a significant increase in [HCO3-]. PaO2 decreased only shortly before death, whereas CaO2 kept elevated due to a large increase in Hct (P < 0.05). We tentatively conclude that cardiac failure is a primary physiological disorder that would lead to death of fish subjected to high environmental CO2 pressures.
机译：在暴露于两种高碳酸血症水平的yellow鱼（Seriola quinqueradiata）中检查了心肺反应。 （用含1％CO2（水PCO2 = 7 mmHg）或5％CO2（38 mmHg）的混合气体平衡的海水）在20℃下放置72小时。在5％CO2的情况下，死亡率在8小时内为100％，而在1％CO2的情况下没有鱼类死亡。暴露于1％CO2期间，未从暴露前的值显着改变心血管变量（点上的心输出量（Q）;心率，HR;中风量，SV和动脉血压，BP）。动脉CO2分压（PaCO2）显着增加（P <0.05），在3小时后达到新的稳态水平。动脉血pH（pHa）最初降低（P <0.05），但随后由于血浆碳酸氢盐（[HCO3-]）升高而恢复。在整个暴露期间，动脉O-2分压（PaO2），氧含量（CaO2）和血细胞比容（Hct）保持不变。相反，尽管HR不变，但暴露于5％CO2的情况下通过降低SV（P <0.05）大大降低了点（P <0.05）（Q）。血压短暂升高（P <0.05），然后在死亡前急剧下降。尽管[HCO3-]显着增加，pHa仍无法完全恢复。 PaO2仅在死亡前不久下降，而CaH2由于Hct的大幅增加而保持升高（P <0.05）。我们初步得出结论，心力衰竭是一种主要的生理疾病，会导致遭受高环境CO2压力的鱼类死亡。

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