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首页> 外文期刊>Journal of cardiovascular electrophysiology >Heart rate turbulence after ventricular and atrial premature beats in subjects without structural heart disease.
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Heart rate turbulence after ventricular and atrial premature beats in subjects without structural heart disease.

机译：在没有结构性心脏病的受试者中，心室和心房过早搏动后的心律紊乱。

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INTRODUCTION: Studies assessing heart rate (HR) behavior after premature beats have focused on HR responses to ventricular premature beats (VBPs), but there is less information of HR behavior after atrial premature beats (APBs). METHODS AND RESULTS: HR turbulence after VPBs and APBs was first measured in response to ambient APBs and VPBs occurring during 24-hour ambulatory ECG recordings in 29 subjects without structural heart disease, and in response to programmed atrial (AE) and ventricular extrastimuli (VE) in 6 subjects undergoing electrophysiologic (EP) examination. Turbulence onset (TO) was more negative (-2.3 +/- 3.2% vs -0.9 +/- 2.8%, P < 0.01) and turbulence slope (TS) was steeper (11 +/- 11 vs 5.1 +/- 4.1 msec/R-R interval, P < 0.05) after VPBs than APBs. Compared to VPBs, the acceleration of HR after APBs was delayed by one beat, and APBs were associated with a short R-R interval preceding the APB, resulting in a blunted TO. Studies of patients undergoing an EP test confirmed the one-beat delay of HR acceleration and the blunted TO after programmed AE compared to VE (P < 0.05). TO and TS after VPBs were related to baroreflex sensitivity. TO also was related to 24-hour standard deviation of N-N intervals (SDNN). However, the TO or TS following APBs was not related to either SDNN or baroreflex sensitivity. CONCLUSION: HR behavior is different in response to APBs and VBPs among subjects without structural heart disease. Different definitions and calculation formulas should be used in the analysis of HR turbulence after APBs and VPBs.
机译：简介：评估早搏后心率（HR）行为的研究集中于对室性早搏（VBP）的HR反应，但对于房性早搏（APB）后的HR行为的信息较少。方法和结果：29位无结构性心脏病的受试者在24小时动态心电图记录期间发生的周围APB和VPB发生时，以及对程序性心房（AE）和心室外刺激（VE）的响应时，首先测量了VPB和APB后的HR湍流）在接受电生理（EP）检查的6位受试者中。湍流起因（TO）更为负面（-2.3 +/- 3.2％vs -0.9 +/- 2.8％，P <0.01）并且湍流斜率（TS）更加陡峭（11 +/- 11 vs 5.1 +/- 4.1毫秒/ RR间隔，P <0.05）。与VPB相比，APB之后的HR加速度延迟了一个心跳，并且APB与APB之前的较短R-R间隔相关，导致TO变钝。对接受EP测试的患者进行的研究证实，与VE相比，程序性AE后HR加速一跳延迟和TO变钝（P <0.05）。 VPB后的TO和TS与压力反射敏感性相关。 TO也与N-N间隔的24小时标准差（SDNN）有关。然而，APB后的TO或TS与SDNN或压力反射敏感性均无关。结论：在无结构性心脏病的受试者中，HR行为对APB和VBP的反应不同。在分析APB和VPB之后的HR湍流时，应使用不同的定义和计算公式。

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