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首页> 外文期刊>Journal of endovascular therapy: an official journal of the International Society of Endovascular Specialists >Magnitude of the forces acting on target vessel stents as a result of a mismatch between native aortic anatomy and fenestrated stent-grafts.
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Magnitude of the forces acting on target vessel stents as a result of a mismatch between native aortic anatomy and fenestrated stent-grafts.

机译：由于天然主动脉解剖结构和有孔支架植入物之间不匹配，作用在目标血管支架上的力的大小。

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PURPOSE: To quantify the compression force acting on target vessel stents as a consequence of the misalignment between the native aortic anatomy and the fenestrated stent-graft owing to measuring errors during the design of the device. METHODS: The material properties of a fenestrated Zenith stent-graft were determined using a standardized tensile testing protocol. Aortic anatomy was modeled using fresh porcine aortas that were subjected to tensile testing. The net force acting on a target vessel stent due to incremental discrepancy between the target vessel ostia and the stent-graft fenestrations was calculated as the difference in wall tension between the aorta and the stent-graft in diastole and systole. The change in diameter between diastole and systole was set to 8%. RESULTS: Using the diastole model, underestimation of circumferential target vessel position by 15 degrees , 22.5 degrees , and 30 degrees resulted in net forces on the target vessel stent of 0.6, 0.8, and 1.1 N, respectively. Overestimation of target vessel position by the same increments resulted in net forces of 0.3, 0.6, and 0.9 N, respectively. With the systolic model, underestimating target vessel position by 30 degrees resulted in a 2.1-N maximum force on the stent, which potentially threatened the seal. In the longitudinal direction, underestimating target vessel separation by up to 10 mm resulted in a maximal force on the stent of 6.1 N, while overestimating target vessel separation did not result in any additional force on the stent due to fabric infolding. CONCLUSION: The magnitude of the forces generated solely due to mismatch between stent-graft design and native anatomy is modest and is unlikely to cause significant deformation of target vessel stents. Mismatch, however, may cause loss of seal.
机译：目的：量化由于在设备设计过程中的测量误差而导致的自然主动脉解剖结构和有孔支架植入物之间未对准的结果，作用在目标血管支架上的压力。方法：使用标准化的拉伸测试方案确定有孔的Zenith支架移植物的材料性能。主动脉解剖模型使用新鲜的猪主动脉进行拉伸测试。计算由于目标血管口和支架移植窗孔之间的增量差异而作用在目标血管支架上的净力，计算为主动脉与支架移植物在舒张期和收缩期之间壁张力的差异。舒张期和收缩期之间的直径变化设为8％。结果：使用舒张模型，将目标血管圆周位置低估15度，22.5度和30度会导致作用在目标血管支架上的净力分别为0.6 N，0.8 N和1.1N。以相同的增量高估目标船只的位置会分别导致0.3、0.6和0.9 N的净力。对于收缩期模型，将目标血管位置低估30度会导致在支架上施加2.1N的最大力，这可能会威胁到密封。在纵向方向上，低估目标血管间距最多10 mm会导致对支架的最大力为6.1 N，而高估目标血管间距不会由于织物的折叠而对支架产生任何额外的力。结论：仅由于支架移植物设计与天然解剖结构不匹配而产生的力大小适中，不太可能导致目标血管支架发生明显变形。但是，不匹配可能会导致密封性丧失。

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