Effect of stress on the production of O(2)(-) in alveolar macrophages.

De Castro-CM; Manh?es de-Castro-R; Fernandes de-Medeiros-A; Queiros Santos-A; Ferreira e-Silva-WT; Luis de-Lima-Filho-J

首页> 外文期刊>Journal of Neuroimmunology: Official Bulletin of the Research Committee on Neuroimmunology of the World Federation of Neurology >Effect of stress on the production of O(2)(-) in alveolar macrophages.

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Effect of stress on the production of O(2)(-) in alveolar macrophages.

机译：应力对肺泡巨噬细胞中O（2）（-）产生的影响。

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Alveolar macrophages (AM) of male rats (200-250 g), stressed or not, were evaluated with relationship to superoxide production (SP). Plasma levels of corticosterone were measured. The control group showed larger SP than the stressed group in all intervals of time. Exposure in vitro of AM to a synthetic glucocorticoid for 40 min (the same time of restraint stress) inhibits SP. Therefore, it seems under stress situations there is an increase of plasma levels of corticosterone and a decrease of SP in AM after stimulation with PMA. O(2)(-) is a potent microbicide and its reduction could cause the loss of microbicidal activity of AM.

机译：评估雄性大鼠（200-250 g）是否受到压力的肺泡巨噬细胞（AM）与超氧化物生成（SP）的关系。测量血浆皮质酮水平。对照组在所有时间间隔内均显示出比压力组更大的SP。在体外将AM暴露于合成糖皮质激素40分钟（同时施加约束压力）会抑制SP。因此，似乎在应激情况下，用PMA刺激后，血浆皮质酮水平增加，而AM中的SP降低。 O（2）（-）是一种有效的杀菌剂，其还原可能导致AM的杀菌活性丧失。

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来源
《Journal of Neuroimmunology: Official Bulletin of the Research Committee on Neuroimmunology of the World Federation of Neurology》 |2000年第2期|共5页
作者
De Castro-CM; Manh?es de-Castro-R; Fernandes de-Medeiros-A; Queiros Santos-A; Ferreira e-Silva-WT; Luis de-Lima-Filho-J;
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正文语种 eng
中图分类神经病学与精神病学;医学免疫学;
关键词
Macrophages; Alveolar; Stress; Superoxides; Tetradecanoylphorbol Acetate; 巨噬细胞; 肺泡; 应激; 超氧化物类;

机译：Macrophages;Alveolar;Stress;Superoxides;Tetradecanoylphorbol Acetate;巨噬细胞;肺泡;应激;超氧化物类;

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Effect of stress on the production of O(2)(-) in alveolar macrophages.

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