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首页> 外文期刊>American Journal of Physiology >Contribution of the respiratory rhythm to sinus arrhythmia in normal unanesthetized subjects during positive-pressure mechanical hyperventilation.
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Contribution of the respiratory rhythm to sinus arrhythmia in normal unanesthetized subjects during positive-pressure mechanical hyperventilation.

机译：在正压机械通气过程中，正常无麻醉对象的呼吸节律对窦性心律不齐的贡献。

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摘要

The precise contribution of the CO2-dependent respiratory rhythm to sinus arrhythmia in eupnea is unclear. The respiratory rhythm and sinus arrhythmia were measured in 12 normal, unanesthetized subjects in normocapnia and hypocapnia during mechanical hyperventilation with positive pressure. In normocapnia (41 +/- 1 mmHg), the respiratory rhythm was always detectable from airway pressure and inspiratory electromyogram activity. The amplitude of sinus arrhythmia (138 +/- 21 ms) during mechanical hyperventilation with positive pressure was not significantly different from that in eupnea. During the same mechanical hyperventilation pattern but in hypocapnia (24 +/- 1 mmHg), the respiratory rhythm was undetectable and the amplitude of sinus arrhythmia was significantly reduced (to 40 +/- 5 ms). These results show a greater contribution to sinus arrhythmia from the respiratory rhythm during hypocapnia caused by mechanical hyperventilation than previously indicated in normal subjects during hypocapnia caused by voluntary hyperventilation. We discuss whether the respiratory rhythm provides the principal contribution to sinus arrhythmia in eupnea.
机译：目前尚不清楚依赖CO2的呼吸节律对呼吸道窦性心律失常的确切作用。在正压机械换气过度期间，在正常碳酸血症和低碳酸血症的12名正常，未麻醉的受试者中测量了呼吸节律和窦性心律不齐。在正常碳酸血症（41 +/- 1 mmHg）中，呼吸节奏通常可通过气道压力和吸气肌电图活动来检测。正压机械通气时窦性心律不齐的幅度（138 +/- 21 ms）与通气时无明显差异。在相同的机械通气过度模式中，但在低碳酸血症（24 +/- 1 mmHg）中，呼吸节律无法检测到，窦性心律不齐的幅度显着降低（降至40 +/- 5 ms）。这些结果表明，机械性过度通气引起的低碳酸血症期间呼吸节律对窦性心律不齐的贡献要大于自愿性过度通气引起的低碳酸血症期间正常受试者对窦性心律不齐的贡献。我们讨论了呼吸节律是否对呼吸道窦性心律不齐提供主要作用。

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