Interleukin-10 limits increased blood pressure and vascular RhoA/Rho-kinase signaling in angiotensin II-infused mice

Lima Victor V.; Zemse Saiprasad M.; Chiao Chin-Wei; Bomfim Gisele F.; Tostes Rita C.; Webb R. Clinton; Giachini Fernanda R.

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Interleukin-10 limits increased blood pressure and vascular RhoA/Rho-kinase signaling in angiotensin II-infused mice

机译：白介素10限制输注血管紧张素II的小鼠的血压升高和血管RhoA / Rho激酶信号传导

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Aims: Interleukin-10 (IL-10) is a multi-functional cytokine with potent anti-inflammatory properties. We hypothesized that IL-10 limits increased RhoA/Rho-kinase signaling and vascular reactivity in arteries from angiotensin II (Ang II) hypertensive mice.

机译：目的：白介素10（IL-10）是一种具有有效抗炎特性的多功能细胞因子。我们假设IL-10限制增加了来自血管紧张素II（Ang II）高血压小鼠动脉的RhoA / Rho激酶信号传导和血管反应性。

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《Life sciences》 |2016年第1期|共7页
作者
Lima Victor V.; Zemse Saiprasad M.; Chiao Chin-Wei; Bomfim Gisele F.; Tostes Rita C.; Webb R. Clinton; Giachini Fernanda R.;
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正文语种 eng
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关键词
Cytokine; Inflammation; Vascular dysfunction;

机译：细胞因子;炎症;血管功能障碍;

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1. Interleukin-10 limits increased blood pressure and vascular RhoA/Rho-kinase signaling in angiotensin II-infused mice [J] . Lima Victor V., Zemse Saiprasad M., Chiao Chin-Wei, Life sciences . 2016,第1期

机译：白介素10限制输注血管紧张素II的小鼠的血压升高和血管RhoA / Rho激酶信号传导
2. Investigation of long chain omega-3 PUFAs on arterial blood pressure, vascular reactivity and survival in angiotensin II-infused Apolipoprotein E-knockout mice [J] . Clinical and experimental pharmacology & physiology . 2016,第2期

机译：长链omega-3 PUFAs对输注血管紧张素II载脂蛋白E基因敲除小鼠的动脉血压，血管反应性和存活率的研究
3. Vascular-targeted overexpression of G protein-coupled receptor kinase-2 in transgenic mice attenuates beta-adrenergic receptor signaling and increases resting blood pressure. [J] . Eckhart AndreaD, Ozaki Tohru, Tevaearai Hendrik, Molecular pharmacology. . 2002,第4期

机译：在转基因小鼠中以血管靶向的G蛋白偶联受体激酶2的过表达减弱了β-肾上腺素受体信号传导并增加了静息血压。
4. Nitric Oxide-Angiotensin II Interaction in Modulation of Vascular Smooth Muscle Signalling: A Possible Role for ROS? [C] . Siobhan Kelleher, Stephen Wilson, Alan K. Keenan NATO Advanced Study Institute on Vascular Endothelium : Mechanisms of Cell Signaling . 1999

机译：一氧化氮 - 血管紧张素II在调节血管平滑肌信号中的相互作用：ROS的可能作用？
5. Increased expression of regulator of G protein signaling 2 explains how choline supplementation increases visceral fat mass in phosphatidylethanolamine N-methyltransferase deficient mice. [D] . Li, Tete. 2011

机译：G蛋白信号转导因子2调节剂的表达增加解释了胆碱补充剂如何增加磷脂酰乙醇胺N-甲基转移酶缺陷型小鼠的内脏脂肪量。
6. Angiotensin II type-1 receptor regulates RhoA and Rho-kinase/ROCK activation via multiple mechanisms. Focus on Angiotensin II induces RhoA activation through SHP2-dependent dephosphorylation of the RhoGAP p190A in vascular smooth muscle cells [O] . Keita Kimura, Satoru Eguchi -1

机译：血管紧张素II 1型受体通过多种机制调节RhoA和Rho激酶/ ROCK活化。专注于血管紧张素II通过依赖SHP2的血管平滑肌细胞中RhoGAP p190A的去磷酸化诱导RhoA激活
7. Interleukin-10 limits increased blood pressure and vascular RhoA/Rho-kinase signaling in angiotensin II-infused mice [O] . Lima Victor V., Zemse Saiprasad M., Chiao Chin-Wei, 2016

机译：白介素10限制了注入血管紧张素II的小鼠的血压升高和血管RhoA / Rho激酶信号传导

Interleukin-10 limits increased blood pressure and vascular RhoA/Rho-kinase signaling in angiotensin II-infused mice

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