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首页> 外文期刊>The Journal of Physiology >Resistance training-induced changes in integrated myofibrillar protein synthesis are related to hypertrophy only after attenuation of muscle damage
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Resistance training-induced changes in integrated myofibrillar protein synthesis are related to hypertrophy only after attenuation of muscle damage

机译:抵抗训练诱导的肌原纤维蛋白合成的综合变化仅在肌肉损伤减弱后才与肥大有关

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摘要

Key points Skeletal muscle hypertrophy is one of the main outcomes from resistance training (RT), but how it is modulated throughout training is still unknown. We show that changes in myofibrillar protein synthesis (MyoPS) after an initial resistance exercise (RE) bout in the first week of RT (T1) were greater than those seen post-RE at the third (T2) and tenth week (T3) of RT, with values being similar at T2 and T3. Muscle damage (Z-band streaming) was the highest during post-RE recovery at T1, lower at T2 and minimal at T3. When muscle damage was the highest, so was the integrated MyoPS (at T1), but neither were related to hypertrophy; however, integrated MyoPS at T2 and T3 were correlated with hypertrophy. We conclude that muscle hypertrophy is the result of accumulated intermittent increases in MyoPS mainly after a progressive attenuation of muscle damage.
机译:要点骨骼肌肥大是抵抗训练(RT)的主要结果之一,但是在整个训练过程中如何调节骨骼肌尚不清楚。我们显示,在RT(T1)的第一周进行首次抵抗运动(RE)发作后,肌原纤维蛋白合成(MyoPS)的变化大于在RE的第三周(T2)和第十周(T3)进行RE后所见的变化。 RT,其值在T2和T3相似。 RE后恢复期间,T1处的肌肉损伤(Z波段流)最高,T2处较低,T3处较小。当肌肉损伤最高时,整合的MyoPS(在T1时)也是如此,但两者均与肥大无关。然而,T2和T3的整合MyoPS与肥大相关。我们得出的结论是,肌肉肥大是主要是在肌肉损伤逐渐减弱之后,MyoPS的累积间歇性增加的结果。

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