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机译:人Cadasil血管平滑肌细胞的自噬 - 溶酶体缺陷
Division of Neurogeriatrics Center for Alzheimer Research Department of Neurobiology Care Sciences and Society;
Division of Neurogeriatrics Center for Alzheimer Research Department of Neurobiology Care Sciences and Society;
Division of Neurogeriatrics Center for Alzheimer Research Department of Neurobiology Care Sciences and Society;
Medicum Biochemistry/Developmental Biology Meilahti Clinical Proteomics Core Facility University of Helsinki;
Division of Neurogeriatrics Center for Alzheimer Research Department of Neurobiology Care Sciences and Society;
Division of Clinical Geriatrics Center for Alzheimer Research Department of Neurobiology Care Sciences and Society;
Division of Neurogeriatrics Center for Alzheimer Research Department of Neurobiology Care Sciences and Society;
Division of Neurogeriatrics Center for Alzheimer Research Department of Neurobiology Care Sciences and Society;
VSMC; LC3; Lysosomes; CADASIL; Autophagy;
机译:人Cadasil血管平滑肌细胞的自噬 - 溶酶体缺陷
机译:胰岛素通过磷脂酰肌醇3激酶和丝裂原激活的蛋白激酶途径激活人和大鼠血管平滑肌细胞中的缺氧诱导因子1α:由于胰岛素信号传导缺陷导致胰岛素抵抗
机译:Hutchinson Gilford早衰症的人iPSC模型揭示了血管平滑肌和间充质干细胞缺陷。
机译:血管平滑肌细胞是从Notch3突变到Cadasil的事件的主要目标
机译:调查使用从脂肪衍生的基质细胞分化的人单核细胞和血管平滑肌样细胞进行血管组织再生
机译:人类光滑肌和血管内皮细胞的直接重新编程揭示了与老化和哈钦森 - 吉尔福德普利亚综合征相关的缺陷
机译:人Cadasil血管平滑肌细胞的自噬 - 溶酶体缺陷