机译:通过Hbub1偏心/泌辛统计组蛋白H2a T120磷酸化的缺陷导致产生多核细胞的染色体误导
Department of BiochemistryNagasaki University School of MedicineNagasaki Japan;
Department of BiochemistryNagasaki University School of MedicineNagasaki Japan;
Department of BiochemistryNagasaki University School of MedicineNagasaki Japan;
Division of Gene Regulation and Signal TransductionSaitama Medical UniversitySaitama Japan;
Department of BiochemistryNagasaki University School of MedicineNagasaki Japan;
Department of BiochemistryNagasaki University School of MedicineNagasaki Japan;
Division of ImmunologyNagasaki UniversityNagasaki Japan;
Division of ImmunologyNagasaki UniversityNagasaki Japan;
Oda ClinicHiroshima Japan;
Division of Gene Regulation and Signal TransductionSaitama Medical UniversitySaitama Japan;
Department of BiochemistryNagasaki University School of MedicineNagasaki Japan;
BUB1; H2A; histone modification; phosphorylation;
机译:通过Hbub1偏心/泌辛统计组蛋白H2a T120磷酸化的缺陷导致产生多核细胞的染色体误导
机译:天然的组蛋白H2A变异体缺少Bub1磷酸化位点,并且对着丝粒组蛋白CENP-A的调节消耗促进了
机译:后期染色体错聚通过组蛋白H3.3 Ser31磷酸化触发p53细胞周期阻滞
机译:在细胞周期中的典型组蛋白H2A磷酸化的自上而下MS / MS分析
机译:酵母ATM和ATR使用不同的机制来围绕DNA双链断裂散布组蛋白H2A磷酸化
机译:天然的组蛋白H2A变异体缺少Bub1磷酸化位点并且在着丝粒白色念珠菌中其着丝粒组蛋白CENP-A的调控耗竭促进了进化
机译:通过Hbub1脱铬/蠕虫大学组蛋白H2a T120磷酸化的缺陷导致产生多核细胞的染色体误导
机译:组蛋白缺陷H3.3磷酸化和aTRX招募到有丝分裂期间错位的染色体有助于小儿胶质母细胞瘤的发展。