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首页> 外文期刊>PACE: Pacing and clinical electrophysiology >Abnormal right atrial electrograms predict the transition to chronic atrial fibrillation in paced patients with sick sinus syndrome.
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Abnormal right atrial electrograms predict the transition to chronic atrial fibrillation in paced patients with sick sinus syndrome.

机译：异常右心房电视图预测病人窦综合征的节奏患者的慢性心房颤动过渡。

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Although pacing therapy for sick sinus syndrome (SSS) is established, the risk of developing chronic atrial fibrillation (CAF) makes pacing therapy infeasible in some patients. We evaluated whether electrophysiological characteristics of atrial muscle can serve as predictors of the transition to CAF after pacemaker implantation in patients with SSS. Eighty-nine patients with SSS underwent electrophysiological study before pacing therapy. Catheter mapping of 12 right atrial sites was performed during sinus rhythm during electrophysiological. An abnormal atrial electrogram was defined as having a duration of 100 ms or longer, or eight or more fragmented deflections, or both. Right atrial extrastimulation was also performed for atrial vulnerability. After electrophysiological study, all patients underwent pacemaker implantation and were followed up. During the follow-up period of 85 +/- 50 months, development of CAF was observed in 12 patients (group A). The remaining 77 patients remained in sinus rhythm (group B). There were significantly more abnormal atrial electrograms in group A than group B (2.7 +/- 2.3 vs 0.8 +/- 1.2; P < 0.001). The distribution of abnormal atrial electrograms was also greater in group A; patients in group A had more abnormal atrial electrograms than patients in group B in both the high and middle right atrium (P < 0.005 and P < 0.01, respectively). Kaplan-Meier analysis showed that almost 50% of the paced patients with abnormal atrial electrograms (n = 42) developed CAF (P < 0.005). Our data suggest that the existence of abnormal atrial electrograms is predictive of the transition to CAF in paced patients with SSS.
机译：尽管建立了病人窦综合征的起搏治疗，但慢性心房颤动（CAF）的风险使起搏治疗在一些患者中不可行。我们评估了心房肌肉的电生理特性是否可以作为SSS患者的起搏器植入后到CAF过渡的预测因子。八十九名SSS患者在起搏治疗前接受了电生理学研究。在电生理过程中，在鼻窦节律期间进行12个右心房部位的导管映射。异常心脏电视图定义为具有100ms或更长的持续时间，或八个或更多个碎裂的偏转，或两者。对心房脆弱性也进行了良好的心房促刺激。电生理学研究后，所有患者都接受了起搏器植入并进行了跟进。在85 +/- 50个月的后续期间，在12名患者（A组）中观察到CAF的发展。剩余的77名患者仍处于窦性心律（B组）。 B组中存在显着的异常心房电视图（2.7 +/- 2.3 Vs 0.8 +/- 1.2; P <0.001）。 A组中异常心脏电视图的分布也更大; A组患者在高中和中右心房（P <0.005和P <0.01）中的B组中的患者具有更异常的心房电视图（分别为P <0.005和P <0.01）。 Kaplan-Meier分析表明，近50％的节奏患者患有异常的心房电视图（n = 42）开发的CAF（P <0.005）。我们的数据表明，存在异常的心房电视图的存在是预测SSS患者的过渡到CAF。

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