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首页> 外文期刊>Protoplasma: An International Journal of Cell Biology >The bacterial volatile dimethyl-hexa-decylamine reveals an antagonistic interaction between jasmonic acid and cytokinin in controlling primary root growth of Arabidopsis seedlings
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The bacterial volatile dimethyl-hexa-decylamine reveals an antagonistic interaction between jasmonic acid and cytokinin in controlling primary root growth of Arabidopsis seedlings

机译：细菌挥发性二甲基 - 六甲胺露出茉莉酸和细胞蛋白之间的拮抗相互作用控制拟南芥幼苗的初级根生长

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摘要

Chemical communication underlies major adaptive traits in plants and shapes the root microbiome. An increasing number of diffusible and/or volatile organic compounds released by bacteria have been identified, which play phytostimulant or protective functions, including dimethyl-hexa-decylamine (DMHDA), a volatile biosynthesized by Arthrobacter agilis UMCV2 that induces jasmonic acid (JA) signaling in Arabidopsis. Here, he found that the growth repressing effects of both DMHDA and JA are antagonized by kinetin and correlated with an inhibition of cytokinin-related ARR5::GUS and TCS::GFP expression in Arabidopsis primary roots. Moreover, we demonstrate that shoot supplementation of JA triggers JAZ1 expression both locally and systemically and represses cytokinin-dependent promoter activity in roots. A similar effect was observed after cotyledon wounding, in which an increase of JA-inducible LOX2:GUS expression represses root growth, which correlates with the loss of TCS::GFP detection at the very root tip. Our data demonstrate that the bacterial volatile DMHDA crosstalks with cytokinin signaling and reveals the downstream antagonistic interaction between JA and cytokinin in controlling root growth.
机译：化学通信在植物中进行主要的适应性性状，并形成根微生物组。已经鉴定了越来越多的细菌释放的扩散和/或挥发性有机化合物，其发挥植物肥胖或保护功能，包括二甲基 - 六甲酰甲胺（DMHDA），由诱导茉莉酸（JA）信号传导的Arthrobacter Agilis UMCV2的挥发性生物合成在拟南芥中。在这里，他发现DMHDA和JA的生长抑制效果被Kinetin拮抗，并与拟南芥初级根中的Cytokinin相关ARR5 :: GUS和TCS :: GFP表达相关。此外，我们证明JA拍摄的补充JA触发JAZ1表达在局部和全身上，并在根中抑制细胞蛋白依赖性启动子活性。在伤口后观察到类似的效果，其中JA-Imucible LOX2：GUS表达的增加抑制了根系生长，其与在非常根尖处的TCS :: GFP检测的损失相关。我们的数据表明，细菌挥发性DMHDA串扰具有细胞蛋白信号传导，并揭示了JA和细胞蛋白之间的下游拮抗相互作用在控制根系生长中。

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