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首页> 外文期刊>Journal of stroke and cerebrovascular diseases: The official journal of National Stroke Association >Endovascular Embolization of a Nondominant Vertebral Artery Compressed by an Osteophyte to Prevent Recurrence of Vertebrobasilar Infarctions
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Endovascular Embolization of a Nondominant Vertebral Artery Compressed by an Osteophyte to Prevent Recurrence of Vertebrobasilar Infarctions

机译：止血剂栓塞骨质体压缩的骨髓椎间动脉，以防止椎体椎间盘复发

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Background: Vertebral artery compression by cervical osteophyte is a rare cause of vertebrobasilar ischemic stroke. This mechanism of stroke has been reported as the Bow Hunter syndrome defined by vertebrobasilar insufficiency because of mechanical stenosis of the vertebral artery at the cervical level triggered by head movement. The most common treatment is surgical decompression. However, in most cases, a dominant vertebral artery is involved, and its dynamic extrinsic compression is demonstrated on angiography. Case report: We report a patient with recurrent posterior circulation infarctions because of the compression of a nondominant vertebral artery by a cervical osteophyte. The dynamic angiography did not show any worsening of the vertebral stenosis by head movements but an irregularity of the vertebral artery with regard to the osteophyte compression, suggesting a direct artery wall injury. We concluded to an embolic mechanism through thrombus formation from the artery wall injury at the stenosed site. Because neither surgical decompression nor stenting was deemed to be a relevant treatment option, endovascular coil embolization of the compressed vertebral artery was performed after a clamping test to check the efficiency of the collateral circulation. The procedure was a success. During the 12-month follow-up, the patient did not have any recurrent stroke. Conclusions: In case of recurrent symptomatic extrinsic compression of a nondominant vertebral artery, endovascular embolization after a clamping test may be considered. (C) 2015 by National Stroke Association
机译：背景：宫颈骨赘的椎动脉压缩是椎弓鼠缺血性卒中的罕见原因。由于头部运动触发的宫颈水平的椎动脉的机械狭窄，脑卒中的这种卒中机制已被报告为椎弓鼠功能不全。最常见的治疗是外科减压。然而，在大多数情况下，涉及主要的椎动脉，并且其动态外在压缩在血管造影上证明。案例报告：由于宫颈骨赘的压缩，我们报告了具有复发后循环动脉的患者。动态血管造影并未显示头部运动的任何恶化，但是椎动脉的不规则性关于骨赘的压缩，表明直接动脉损伤。我们通过在狭窄的部位在动脉壁损伤中形成栓塞机制。由于外科减压和支架都认为是相关的治疗方法，因此在夹紧试验后进行压缩椎动脉的血管内线圈栓塞，以检查侧支循环的效率。该程序取得了成功。在12个月的随访期间，患者没有任何复发性卒中。结论：在夹紧试验后复发性症状外在压缩的情况下，可以考虑夹紧试验后的血管内栓塞。 （c）2015年国家冲程协会

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