Resolution of atopic dermatitis (AD) transcriptomics with an in silico CIBERSORT algorithm reveals that interleukin (IL)-4-IL-13 modify keratinocytes in both lesional and nonlesional skin, whereas an IL-17 signature distinguishes chronic AD

Clayton K.; Davies J.; Sirvent S.; Vallejo A.; Ginger R.; Lim F. -L.; Ardern-Jones M.; Polak M.

首页> 外文期刊>British Journal of Dermatology >Resolution of atopic dermatitis (AD) transcriptomics with an in silico CIBERSORT algorithm reveals that interleukin (IL)-4-IL-13 modify keratinocytes in both lesional and nonlesional skin, whereas an IL-17 signature distinguishes chronic AD

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Resolution of atopic dermatitis (AD) transcriptomics with an in silico CIBERSORT algorithm reveals that interleukin (IL)-4-IL-13 modify keratinocytes in both lesional and nonlesional skin, whereas an IL-17 signature distinguishes chronic AD

机译：分辨率的特应性皮炎（Ad）转录组织用硅纤维区算法表明，白细胞介素（IL）-4-IL-13在裂变和无缘皮肤中修饰角质形成细胞，而IL-17签名区分慢性广告

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来源
《British Journal of Dermatology》 |2019年第6期|共2页
作者
Clayton K.; Davies J.; Sirvent S.; Vallejo A.; Ginger R.; Lim F. -L.; Ardern-Jones M.; Polak M.;
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Univ Southampton Southampton Hants England;

Univ Southampton Southampton Hants England;

Univ Southampton Southampton Hants England;

Univ Southampton Southampton Hants England;

Unilever London England;

Unilever London England;

Univ Southampton Southampton Hants England;

Univ Southampton Southampton Hants England;

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正文语种 eng
中图分类皮肤病学与性病学;
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1. Resolution of atopic dermatitis (AD) transcriptomics with an in silico CIBERSORT algorithm reveals that interleukin (IL)-4-IL-13 modify keratinocytes in both lesional and nonlesional skin, whereas an IL-17 signature distinguishes chronic AD [J] . Clayton K., Davies J., Sirvent S., British Journal of Dermatology . 2019,第6期

机译：分辨率的特应性皮炎（Ad）转录组织用硅纤维区算法表明，白细胞介素（IL）-4-IL-13在裂变和无缘皮肤中修饰角质形成细胞，而IL-17签名区分慢性广告
2. Biophysical characteristics of healthy skin and nonlesional skin in atopic dermatitis: short-term effects of ultraviolet A and B irradiation. [J] . Habig J, Vocks E, Kautzky F, Skin pharmacology and applied skin physiology . 2000,第3a4期

机译：特应性皮炎中健康皮肤和非病变皮肤的生物物理特性：紫外线A和B辐射的短期影响。
3. Topical administration of reversible SAHH inhibitor ameliorates imiquimod-induced psoriasis-like skin lesions in mice via suppression of TNF-alpha/IFN-gamma-induced inflammatory response in keratinocytes and T cell-derived IL-17 [J] . Lin Ze-Min, Ma Meng, Li Heng, Pharmacological research: The official journal of The Italian Pharmacological Society . 2018,第期

机译：局部施用可逆织甲抑制剂通过抑制角质形成细胞和T细胞衍生的IL-17中的TNF-α/ IFN-Gamma诱导的TNF-α/ IFN-Gamma诱导的炎症反应来改善咪喹莫特诱导的伊替菊酯状皮肤病变。
4. CD8+ T cells in the lesional skin of atopic dermatitis and psoriasis patients are an important source of IFN-γ IL-13 IL-17 and IL-22 [O] . DirkJan Hijnen, Edward F. Knol, Yoony Y. Gent, -1

机译：CD8 + T细胞中的特应性皮炎和牛皮癣患者的损伤皮肤是IFN-γ的一个重要来源IL-13IL-17和IL-22
5. Nonlesional atopic dermatitis skin is characterized by broad terminal differentiation defects and variable immune abnormalities. [O] . Suárez-Fariñas M, Tintle SJ, Shemer A, 2011

机译：非损伤性特应性皮炎皮肤的特征在于广泛的终末分化缺陷和可变的免疫异常。

Resolution of atopic dermatitis (AD) transcriptomics with an in silico CIBERSORT algorithm reveals that interleukin (IL)-4-IL-13 modify keratinocytes in both lesional and nonlesional skin, whereas an IL-17 signature distinguishes chronic AD

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