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首页> 外文期刊>Current medicinal chemistry-Anti-inflammatory & anti-allergy agents.Anti-inflammatory & anti-allergy agents >Innate Immunity and Cytokines: How to Regulate Host Response Against Pathogens Avoiding Autoreactivity
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Innate Immunity and Cytokines: How to Regulate Host Response Against Pathogens Avoiding Autoreactivity

机译：先天免疫和细胞因子:如何监管避免对病原体的宿主反应Autoreactivity

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摘要

From the immunological point of view, health can be defined as the results of the balance between host response against invading pathogens (bacteria, virures, fungi, parasites) and the control of autoimmune reactions. In particular, the immune response must undergo several passages involving different cell types, such as the effectors of the so-called natural or innate immunity (monocytes, granulocytes and natural killer, NK, cells), able to start the fight against foreign invaders, and the effclors of acquired immunity, such as alphabeta and gammadelta T lymphocytes, responsible for a more selective, antigen-specific reaction. In both cases the response must be self-limited, in order to avoid or limit tissue damage due to inflammation and/or autoimmunity. The correct progression of immuno-inflammatory responses depends on different intercellular communications for which eytokines function as major messengers. In this issue many aspects of this problem will be presented, from their involvement in the pathogenesis of infectious and autoimmune diseases to their potential role in diagnosis and therapy.
机译：从免疫学的角度来看,健康被定义为之间的平衡的结果主机响应对入侵的病原体(virures细菌、真菌、寄生虫)自体免疫反应的控制。免疫反应必须经历几个段落涉及不同的细胞类型,如效应器所谓的自然或天生的免疫(单核细胞、粒细胞和自然杀手,NK细胞),能够开始战斗对外国侵略者,effclors后天免疫,如alphabeta和gammadelta T淋巴细胞,更负责选择性,抗原特异反应。情况下,响应必须是自限性的,为了避免或限制导致组织损伤炎症和/或自身免疫。immuno-inflammatory反应的进展取决于不同的细胞间通讯eytokines函数作为主要的使者。在这个问题的许多方面这个问题了,从他们的参与传染病和自体免疫发病机制在诊断和疾病的潜在作用治疗。

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