The management of neonatal pulmonary hypertension.

Rami Dhillon

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The management of neonatal pulmonary hypertension.

机译：新生儿肺动脉高压的管理。

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Most neonates with clinically significant pulmonary hypertension (PH) will have either persistent PH of the newborn (PPHN) or bronchopulmonary dysplasia. Cyanotic congenital heart disease must be actively ruled out as part of the differential diagnosis of PPHN. The maintenance of ductal patency with prostaglandins E1 or E2 in cases of doubt is safe and potentially beneficial given their pulmonary vasorelaxant properties. Specific tools in the treatment of PPHN include modern ventilatory strategies, inhaled nitric oxide, sildenafil, prostacyclin and extracorporeal membrane oxygenation. Rarely will a cardiac lesion be primarily responsible for neonatal PH although pulmonary vein stenosis and the persistence of an arterial duct must be considered, particularly in the older preterm baby with bronchopulmonary dysplasia.

机译：大多数具有临床显着性肺动脉高压（PH）的新生儿将具有持续性新生儿PH（PPHN）或支气管肺发育不良。作为PPHN鉴别诊断的一部分，必须积极排除先天性紫癜性心脏病。如果存在疑问，使用前列腺素E1或E2维持导管通畅是安全的，并且鉴于其肺血管舒张特性，可能会有益。治疗PPHN的具体工具包括现代通气策略，吸入一氧化氮，西地那非，前列环素和体外膜氧合。尽管必须考虑肺静脉狭窄和动脉导管的持久性，但心脏病变很少是新生儿PH的主要原因，特别是在患有支气管肺发育不良的大龄早产婴儿中。

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《Archives of disease in childhood. Fetal and neonatal edition》 |2012年第3期|共6页
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Rami Dhillon;
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1. The management of neonatal pulmonary hypertension. [J] . Rami Dhillon Archives of disease in childhood. Fetal and neonatal edition . 2012,第3期

机译：新生儿肺动脉高压的管理。
2. Cinaciguat, a soluble guanylate cyclase activator, augments cGMP after oxidative stress and causes pulmonary vasodilation in neonatal pulmonary hypertension. [J] . Chester M, Seedorf G, Tourneux P, American Journal of Physiology . 2011,第5Parta1期

机译：Cinaciguat，一种可溶性胍基环酶活化剂，氧化应激后CGMP增强，并导致新生儿肺动脉高压肺血管舒张。
3. Cinaciguat, a soluble guanylate cyclase activator, augments cGMP after oxidative stress and causes pulmonary vasodilation in neonatal pulmonary hypertension. [J] . Chester M, Seedorf G, Tourneux P, American Journal of Physiology . 2011,第5Parta1期

机译：Cinaciguat，一种可溶性胍基环酶活化剂，氧化应激后CGMP增强，并导致新生儿肺动脉高压肺血管舒张。
4. Inhaled nitric oxide in preterm and term neonates with hypoxic respiratoty failure and persistent pulmonary hypertension. [C] . Acta pharmacologica sinica . 2002

机译：患有低氧性呼吸衰竭和持续性肺动脉高压的早产和足月新生儿吸入一氧化氮。
5. Dynamic system models to noninvasively predict pulmonary vascular stiffness, pulmonary vascular resistance, flow and heart work in children with pulmonary hypertension. [D] . Gross, Justin K. 2007

机译：动态系统模型可无创地预测肺动脉高压儿童的肺血管僵硬度，肺血管阻力，血流和心脏功能。
6. Persistence re-expression and induction of pulmonary arterial fibronectin tropoelastin and type I procollagen mRNA expression in neonatal hypoxic pulmonary hypertension. [O] . A. G. Durmowicz, W. C. Parks, D. M. Hyde, 1994

机译：新生儿缺氧性肺动脉高压中肺动脉纤连蛋白原弹性蛋白和I型胶原蛋白mRNA的持续性再表达和诱导。
7. Persistence of the fetal pattern of tropoelastin gene expression in severe neonatal bovine pulmonary hypertension. [O] . Stenmark, K R, Durmowicz, A G, Roby, J D, 1994

机译：胎儿原弹性蛋白基因表达的胎儿模式在严重新生儿牛肺动脉高压中的持久性。

The management of neonatal pulmonary hypertension.

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