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首页> 外文期刊>International Journal of Zoology >Glycolipids as Potential Energy Molecules during Starvation in Climbing Perch, Anabas testudineus (Bloch)
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Glycolipids as Potential Energy Molecules during Starvation in Climbing Perch, Anabas testudineus (Bloch)

机译：糖脂是饥饿状态下Anaphas testudineus（Bloch）攀缘鲈鱼的潜在能量分子

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摘要

Glycolipids are membrane lipids which act as cellular markers and also provide energy for the cells. The present study is an attempt to understand whether glycolipids can act as energy sources during fasting. To achieve this, we selected and subjectedAnabas testudineus to short-term (15 days) and long-term (60 days) laboratory starvation. We estimated glycolipids biochemically using a standard protocol in six different tissues. Results showed a selective decline in glycolipid concentration in certain tissues, and also an increase was observed in some tissues. Short-term fasting led to a decline in glycolipids in tissues such as brain (P < 0.05), accessory respiratory organ (P < 0.001), pectoral and lateral line muscle. Liver and kidney (P < 0.002)reported an increase. Long term starvation also resulted in a decline in tissues such as liver (P < 0.001), kidney (P < 0.001), brain, and accessory respiratory organ. Muscle tissue, that is, both the pectoral (P < 0.002) and lateral line muscle (P < 0.05), showed an increase in the glycolipid fraction. This selective decline in glycolipid content of certain tissues suggests a possible utilization of these lipids during starvation and the significant upsurge observed in certain tissues suggests a simultaneous synthesis occurring along the degradation, probably reducing the oxidative stress created by ROS (reactive oxygen species).
机译：糖脂是膜脂质，其充当细胞标记并且还为细胞提供能量。本研究试图了解糖脂在禁食期间是否可以充当能量来源。为了达到这个目的，我们选择了Anabas testudineus进行短期（15天）和长期（60天）实验室饥饿。我们使用标准方案在六个不同组织中生化评估糖脂。结果显示某些组织中的糖脂浓度选择性降低，并且在某些组织中也观察到升高。短期禁食可导致脑（P <0.05），副呼吸器官（P <0.001），胸肌和侧线肌等组织中的糖脂减少。肝和肾（P <0.002）报告有所增加。长期饥饿还导致肝脏（P <0.001），肾脏（P <0.001），大脑和呼吸器官副器官的组织减少。肌肉组织，即胸肌（P <0.002）和侧线肌（P <0.05）均显示糖脂分数增加。某些组织中糖脂含量的这种选择性下降表明在饥饿期间可能利用了这些脂质，并且在某些组织中观察到的显着升高表明降解过程中同时发生了合成，可能减少了由ROS（活性氧）产生的氧化应激。

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