Macular edema (ME) is the major cause of visual impairment in uveitis.1 ME develops because of a breakdown of the inner and/or outer blood retinal barrier caused by inflammation, followed by an influx of proteins and fluid from the plasma. ME may persist even after the inflammation has subsided due to a permanently compromised blood retinal barrier. Uveitis entities frequently associated with ME include sarcoidosis-associated uveitis and birdshot chorioretinopathy. Macular thickening on optical coherence tomography (OCT) was found in 42% of 43 unilateral acute anterior uveitis compared with the normal fellow eye. The presence of ME is linked to the visual outcome of uveitis, and all cases irrespective of their concurrent visual acuity should be treated before the definitive structural changes have set in. Herein, we summarize recent developments regarding the use of imaging techniques for inflammatory ME.
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