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首页> 外文期刊>Medical engineering & physics. >Biomechanical responses due to discitis infection of a juvenile thoracolumbar spine using finite element modeling
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Biomechanical responses due to discitis infection of a juvenile thoracolumbar spine using finite element modeling

机译：少年胸腰椎椎间盘炎感染引起的生物力学响应的有限元建模

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摘要

Growth modulation changes occur in pediatric spines and lead to kyphotic deformity during discitis infection from mechanical forces. The present study was done to understand the consequences of discitis by simulating inflammatory puss at the T12/L1 disc space using a validated eight-year-old thoracolumbar spine finite element model. Changes in the biomechanical responses of the bone, disc and ligaments were determined under physiological compression and flexion loads in the intact and discitis models. During flexion, the angular-displacement increased by 3.33 times the intact spine and localized at the infected junction (IJ). The IJ became a virtual hinge. During compression loading, higher stresses occurred in the growth plate superior to the IJ. The components of the principal stresses in the growth plates at the T12/L1 junction indicated differential stresses. The strain increased by 143% during flexion loading in the posterior ligaments. The study indicates that the flexible pediatric spine increases the motion of the infected spine during physiological loadings. Understanding intrinsic responses around growth plates is important within the context of growth modulation in children. These results are clinically relevant as it might help surgeons to come up with better decisions while developing treatment protocols or performing surgeries.
机译：小儿脊柱发生生长调节变化，并在机械力引起的椎间盘炎感染期间导致后凸畸形。本研究是通过使用经过验证的八岁胸腰椎脊柱有限元模型模拟T12 / L1椎间盘间隙的炎性脓疮来了解椎间盘炎的后果的。在完整的和椎间盘炎模型中，在生理压缩和屈曲负荷下确定了骨骼，椎间盘和韧带的生物力学响应的变化。在屈曲过程中，角位移增加了完整脊柱的3.33倍，并位于受感染的交界处（IJ）。 IJ变成了虚拟铰链。在压缩加载过程中，生长板上的应力高于IJ。在T12 / L1交界处的生长板中的主应力分量表明存在差异应力。在后韧带屈曲加载过程中，应变增加了143％。研究表明，柔性小儿脊柱在生理负荷期间会增加受感染脊柱的运动。在儿童生长调节的背景下，了解生长板周围的内在反应很重要。这些结果在临床上具有相关性，因为这可能有助于外科医生在制定治疗方案或进行手术时做出更好的决定。

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