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首页> 外文期刊>Kleintier-Praxis: Archiv fur Kleine Haus- und Nutztiere Sowie Laboratoriums- und Zoo-Tiere >A case of mitochondrial myopathy in a four-month-old mixed-breed dog. [German]Original Title Mitochondriale Myopathic bei einem vier Monate alten Schnauzer-Mix.
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A case of mitochondrial myopathy in a four-month-old mixed-breed dog. [German]Original Title Mitochondriale Myopathic bei einem vier Monate alten Schnauzer-Mix.

机译：四个月大的混血狗的线粒体肌病一例。 [德语] Monate alten Schnauzer-Mix原版线粒体肌病。

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摘要

A four-month-old Schnauzer-mix was presented with an exercise intolerance. The clinical examination revealed evidence of mitochondrial myopathy. Thoracic radiographs were found to be normal. Electromyography was also within normal limits. Muscle biopsies were taken and showed an alteration of size and structure in the mitochondria. A decrease in oxidative activity was noted. These are histopathological signs for unspecific mitochondrial myopathy. Such damage in the mitochondria leads to a loss of enzyme activity. Furthermore, it leads to a loss of oxidative phosphorylation and energy in the muscle cells. Obviously, the dog's exercise intolerance was due to the energy loss in its muscle cells. So far the aetiology of this disease is unknown; therefore, conservative treatment with exercise restriction was chosen. Six months after he was first presented, the patient still showed episodes of weakness lasting for a few minutes; however, most of the time the patient remained free of symptoms.
机译：出现了一个四个月大的雪纳瑞混合饲料，运动不耐症。临床检查显示线粒体肌病的证据。发现胸部X光片正常。肌电图也处于正常范围内。进行肌肉活检，并显示线粒体的大小和结构发生了变化。注意到氧化活性降低。这些是非特异性线粒体肌病的组织病理学迹象。线粒体中的这种破坏导致酶活性的损失。此外，它导致肌肉细胞中氧化磷酸化和能量的损失。显然，狗的运动不耐受是由于其肌肉细胞的能量损失所致。到目前为止，该病的病因尚不清楚。因此，选择运动受限的保守治疗。首次就诊六个月后，患者仍然表现出持续几分钟的无力发作。但是，大多数情况下，患者没有症状。

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