Induction of Bcl11b during T cell commitment through a tripartite mechanism

Georgopoulos Katia

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Induction of Bcl11b during T cell commitment through a tripartite mechanism

机译：通过三方机制在T细胞定殖过程中诱导Bcl11b

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Induction of the transcription factor Bcl11b in early T cell precursors is supported by three positive forces: signaling via the transmembrane receptor Notch1 provides stochastic permissivity; the transcription factors TCF-1 and GATA-3 poise the locus for expression; and the transcription factor Runx1 steps in to raise expression.

机译：早期T细胞前体中转录因子Bcl11b的诱导受到三个正向作用的支持：通过跨膜受体Notch1的信号提供随机的介导性;转录因子TCF-1和GATA-3处于表达位点。转录因子Runx1参与表达。

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《Nature immunology》 |2016年第8期|共2页
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Georgopoulos Katia;
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正文语种 eng
中图分类医学免疫学;
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1. Induction of Bcl11b during T cell commitment through a tripartite mechanism [J] . Georgopoulos Katia Nature immunology . 2016,第8期

机译：通过三方机制在T细胞定殖过程中诱导Bcl11b
2. Mathematical modelling of interacting mechanisms for hypoxia mediated cell cycle commitment for mesenchymal stromal cells [J] . Bo Zhang, Hua Ye, Aidong Yang BMC Systems Biology . 2018,第1期

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5. Bcl11b, a t-cell commitment factor, and its role in human immunodeficiency virus-1 transcription [D] . Woerner, Andrew J. 2014

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机译：四个调节因子的异步组合作用激活Bcl11b的T细胞承诺
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Induction of Bcl11b during T cell commitment through a tripartite mechanism

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