...
  • 主题
高级搜索  >
历史搜索 清空历史
首页> 外文期刊>Cardiovascular and Interventional Radiology: A Journal of Imaging in Diagnosis and Treatment >Intrahepatic left to right portoportal venous collateral vascular formation in patients undergoing right portal vein ligation
【24h】

Intrahepatic left to right portoportal venous collateral vascular formation in patients undergoing right portal vein ligation

机译：右门静脉结扎患者肝内左至右门静脉静脉侧支血管形成

获取原文
获取原文并翻译 | 示例
           
页面导航
  • 摘要
  • 著录项
  • 相似文献
  • 相关主题

摘要

Purpose: We investigated intrahepatic vascular changes in patients undergoing right portal vein ligation (PVL) or portal vein embolization (PVE) in conjunction with the ensuing hypertrophic response and function of the left liver lobe. Methods: Between December 2008 and October 2011, 7 patients underwent right PVL and 14 patients PVE. Computed tomographic (CT) volumetry to assess future remnant liver (FRL) and functional hepatobiliary scintigraphy were performed in all patients before and 3 weeks after portal vein occlusion. In 18 patients an intraoperative portography was performed to assess perfusion through the occluded portal branches. Results: In all patients after initially successful PVL, reperfused portal veins were observed on CT scan 3 weeks after portal occlusion. This was confirmed in all cases during intraoperative portography. Intrahepatic portoportal collaterals were identified in all patients in the PVL group and in one patient in the PVE group. In all other PVE patients, complete occlusion of the embolized portal branches was observed on CT scan and on intraoperative portography. The median increase of FRL volume after PVE was 41.6 % (range 10-305 %), and after PVL was only 8.1 % (range 0-102 %) (p = 0.179). There were no differences in FRL function between both groups. Conclusion: Preoperative PVE and PVL are both methods to induce hypertrophy of the FRL in anticipation of major liver resection. Compared to PVE, PVL seems less efficient in inducing hypertrophy of the nonoccluded left lobe. This could be caused by the formation of intrahepatic portoportal neocollateral vessels, through which the ligated portal branches are reperfused within 3 weeks.
机译：目的：我们调查了右门静脉结扎（PVL）或门静脉栓塞（PVE）以及随之而来的左肝叶肥大反应和功能的患者的肝内血管变化。方法：在2008年12月至2011年10月之间，有7例患者接受了右PVL治疗，有14例患者接受了PVE治疗。在门静脉阻塞之前和之后3周，对所有患者进行了计算机断层扫描（CT）量度，以评估未来的残余肝脏（FRL）和功能性肝胆闪烁显像。在18例患者中，术中进行了门静脉造影，以评估通过闭塞的门静脉分支的灌注情况。结果：在最初成功实施PVL后的所有患者中，在门静脉闭塞3周后的CT扫描中观察到了再灌注的门静脉。术中门静脉造影期间所有病例均证实了这一点。在PVL组的所有患者和PVE组的一名患者中均发现了肝内门脉侧支。在所有其他PVE患者中，在CT扫描和术中术中均观察到栓塞的门静脉分支完全闭塞。 PVE后FRL体积的中位数增加为41.6％（范围为10-305％），而PVL后仅为8.1％（范围为0-102％）（p = 0.179）。两组之间的FRL功能没有差异。结论：术前PVE和PVL都是导致大面积肝切除的FRL肥大的方法。与PVE相比，PVL诱导非阻塞性左叶肥大的效率似乎较低。这可能是由于肝内门静脉新侧支血管的形成引起的，结扎的门静脉分支通过这些血管在3周内重新灌注。

著录项

相似文献

  • 外文文献
  • 中文文献
  • 专利
获取原文

客服邮箱：kefu@zhangqiaokeyan.com

京公网安备：11010802029741号 ICP备案号：京ICP备15016152号-6 六维联合信息科技 (北京) 有限公司©版权所有

  • 客服微信

  • 服务号